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Clinical patterns and biological correlates of cognitive dysfunction associated with cancer therapy Dietrich J; Monje M; Wefel J; Meyers COncologist 2008[Dec]; 13 (12): 1285-95Standard oncological therapies, such as chemotherapy and cranial radiotherapy, frequently result in a spectrum of neurocognitive deficits that includes impaired learning, memory, attention, and speed of information processing. In addition to classical mechanisms of neurotoxicity associated with chemo- and radiotherapy, such as radiation necrosis and leukoencephalopathy, damage to dynamic progenitor cell populations in the brain is emerging as an important etiologic factor. Radiation- and chemotherapy-induced damage to progenitor populations responsible for maintenance of white matter integrity and adult hippocampal neurogenesis is now believed to play a major role in the neurocognitive impairment many cancer survivors experience.|Antineoplastic Agents/*adverse effects[MESH]|Cognition Disorders/*etiology/prevention & control[MESH]|Hippocampus/radiation effects[MESH]|Humans[MESH]|Magnetic Resonance Imaging[MESH]|Neoplasms/psychology/*therapy[MESH]|Neurons/drug effects/radiation effects[MESH]|Radiotherapy/*adverse effects[MESH]|Stem Cells/drug effects/radiation effects[MESH] |
