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10.1016/bs.vh.2018.10.004 http://scihub22266oqcxt.onion/10.1016/bs.vh.2018.10.004 30678852!ä!30678852 Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=30678852&cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215 Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\30678852.jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       Vitam+Horm 2019 ; 109 (ä): 105-131 Nephropedia Template TP {{tp|p=30678852|t=2019. Aldosterone and Ion Channels |pdf=|usr=}}{{30678852}} gab.com Text Aldosterone and Ion Channels JO: Vitam Horm http://www.kidney.de/pget.php?&tw=1&pmid=30678852 #FOAvip Since its discovery, aldosterone and ion modulation have been entwined. While scientific investigations throughout the decades have emphasized aldosterone's connection to Na(+), K(+), and H(+) homeostasis, more recent research has demonstrated a relationship between aldosterone and Mg(2+), Ca(2+), and Cl(-) homeostasis. The mechanisms connecting aldosterone to ion regulation frequently involve ion channels; the membrane localized proteins containing at least one aqueous pore for ion conduction. In order to precisely control intracellular or intraorganelle ion concentrations, ion channels have evolved highly specific regions within the conduction pore that select ions by charge, size, and/or dehydration energy requirement, meaning aldosterone must be able to modulate multiple ion channels to regulate the many ions described above. The list of ion channels presently connected to aldosterone includes ENaC (Na(+)), ROMK/BK (K(+)), TRPV4/5/6 (Ca(2+)), TRPM7/6 (Mg(2+)), and ClC-K/CFTR (Cl(-)), among others. This list is only expected to grow over time, as the promiscuity of aldosterone becomes more understood. Twit Text FOAVip Aldosterone and Ion Channels ????Vitam Horm http://www.kidney.de/pget.php?&tw=1&pmid=30678852 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=30678852 #FOAvip English Wikipedia <ref>{{Cite pmid|30678852}}</ref> Aldosterone and Ion Channels #MMPMID30678852 Valinsky WC; Touyz RM; Shrier A Vitam Horm 2019[]; 109 (ä): 105-131 PMID30678852show ga Since its discovery, aldosterone and ion modulation have been entwined. While scientific investigations throughout the decades have emphasized aldosterone's connection to Na(+), K(+), and H(+) homeostasis, more recent research has demonstrated a relationship between aldosterone and Mg(2+), Ca(2+), and Cl(-) homeostasis. The mechanisms connecting aldosterone to ion regulation frequently involve ion channels; the membrane localized proteins containing at least one aqueous pore for ion conduction. In order to precisely control intracellular or intraorganelle ion concentrations, ion channels have evolved highly specific regions within the conduction pore that select ions by charge, size, and/or dehydration energy requirement, meaning aldosterone must be able to modulate multiple ion channels to regulate the many ions described above. The list of ion channels presently connected to aldosterone includes ENaC (Na(+)), ROMK/BK (K(+)), TRPV4/5/6 (Ca(2+)), TRPM7/6 (Mg(2+)), and ClC-K/CFTR (Cl(-)), among others. This list is only expected to grow over time, as the promiscuity of aldosterone becomes more understood. |Aldosterone/*metabolism[MESH] |Animals[MESH] |Gene Expression Regulation/*physiology[MESH] |Genomics[MESH]Aldosterone and Ion Channels (epmc).pdf DeepDyve Pubget Overpricing