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10.1016/j.mcna.2016.09.005 http://scihub22266oqcxt.onion/10.1016/j.mcna.2016.09.005 C5308549!5308549!28189171     free     free     free Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\28189171.jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       Med+Clin+North+Am 2017 ; 101 (2): 285-96 Nephropedia Template TP {{tp|p=28189171|t=2017. Anemia of Inflammation: A Review |pdf=|usr=}}{{28189171}} gab.com Text Anemia of Inflammation: A Review JO: Med Clin North Am http://www.kidney.de/pget.php?&tw=1&pmid=28189171 #FOAvip Impaired iron homeostasis and the suppressive effects of proinflammatory cytokines on erythropoiesis, together with alterations of the erythrocyte membrane that impair its survival, cause the anemia of inflammation. Recent epidemiologic studies have connected inflammatory anemia with critical illness, obesity, aging, and kidney failure, as well as with cancer, chronic infection, and autoimmune disease. The proinflammatory cytokine, interleukin-6, the iron regulatory hormone, hepcidin, and the iron exporter, ferroportin, interact to cause iron sequestration in the setting of inflammation. While severe anemia is associated with adverse outcomes in critical illness, experimental models suggest that iron sequestration is part of a natural defense against pathogens. In animal models and human patients, experimental therapeutic approaches targeting interleukin-6 or the ferroportin-hepcidin axis have shown efficacy in reversing anemia, although these agents have not yet been approved for the treatment of the anemia of inflammation. Twit Text FOAVip Anemia of Inflammation: A Review ????Med Clin North Am http://www.kidney.de/pget.php?&tw=1&pmid=28189171 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=28189171 #FOAvip English Wikipedia <ref>{{Cite pmid|28189171}}</ref> Anemia of Inflammation: A Review #MMPMID28189171 Fraenkel PG Med Clin North Am 2017[Mar]; 101 (2): 285-96 PMID28189171show ga Impaired iron homeostasis and the suppressive effects of proinflammatory cytokines on erythropoiesis, together with alterations of the erythrocyte membrane that impair its survival, cause the anemia of inflammation. Recent epidemiologic studies have connected inflammatory anemia with critical illness, obesity, aging, and kidney failure, as well as with cancer, chronic infection, and autoimmune disease. The proinflammatory cytokine, interleukin-6, the iron regulatory hormone, hepcidin, and the iron exporter, ferroportin, interact to cause iron sequestration in the setting of inflammation. While severe anemia is associated with adverse outcomes in critical illness, experimental models suggest that iron sequestration is part of a natural defense against pathogens. In animal models and human patients, experimental therapeutic approaches targeting interleukin-6 or the ferroportin-hepcidin axis have shown efficacy in reversing anemia, although these agents have not yet been approved for the treatment of the anemia of inflammation. äAnemia of Inflammation: A Review (epmc).pdf DeepDyve Pubget Overpricing