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10.1172/JCI80818 http://scihub22266oqcxt.onion/10.1172/JCI80818 C4362244!4362244!25689262     free     free     free Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\25689262.jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       J+Clin+Invest 2015 ; 125 (3): 972-4 Nephropedia Template TP {{tp|p=25689262|t=2015. Benign hyperfiltration after living kidney donation |pdf=|usr=}}{{25689262}} gab.com Text Benign hyperfiltration after living kidney donation JO: J Clin Invest http://www.kidney.de/pget.php?&tw=1&pmid=25689262 #FOAvip Almost one-third of transplanted kidneys come from living donors, who sacrifice approximately 30% of their pre-donation glomerular filtration rate (GFR) after they experience compensatory hypertrophy and hyperfiltration in their remaining kidney. Although hyperfiltration can cause glomerular injury, many studies have suggested that donor nephrectomy itself does not cause long-term loss of GFR at a higher rate than what is seen in the normal aging population. However, when post-donation kidney diseases occur in an unfortunate few, recent studies suggest that GFR loss at donor nephrectomy increases the risk of eventual end-stage renal disease (ESRD). In this issue of the JCI, Lenihan and colleagues evaluated glomerular dynamics in a cohort of kidney donors prior to, within 1 year of, and several years after kidney donation. Their results suggest that adaptive hyperfiltration in the remaining kidney occurs without glomerular hypertension, furthering our understanding of the relatively benign renal outcomes for most living kidney donors. Twit Text FOAVip Benign hyperfiltration after living kidney donation ????J Clin Invest http://www.kidney.de/pget.php?&tw=1&pmid=25689262 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=25689262 #FOAvip English Wikipedia <ref>{{Cite pmid|25689262}}</ref> Benign hyperfiltration after living kidney donation #MMPMID25689262 Blantz RC; Steiner RW J Clin Invest 2015[Mar]; 125 (3): 972-4 PMID25689262show ga Almost one-third of transplanted kidneys come from living donors, who sacrifice approximately 30% of their pre-donation glomerular filtration rate (GFR) after they experience compensatory hypertrophy and hyperfiltration in their remaining kidney. Although hyperfiltration can cause glomerular injury, many studies have suggested that donor nephrectomy itself does not cause long-term loss of GFR at a higher rate than what is seen in the normal aging population. However, when post-donation kidney diseases occur in an unfortunate few, recent studies suggest that GFR loss at donor nephrectomy increases the risk of eventual end-stage renal disease (ESRD). In this issue of the JCI, Lenihan and colleagues evaluated glomerular dynamics in a cohort of kidney donors prior to, within 1 year of, and several years after kidney donation. Their results suggest that adaptive hyperfiltration in the remaining kidney occurs without glomerular hypertension, furthering our understanding of the relatively benign renal outcomes for most living kidney donors. äBenign hyperfiltration after living kidney donation (epmc).pdf DeepDyve Pubget Overpricing