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10.3389/fimmu.2018.00838 http://scihub22266oqcxt.onion/10.3389/fimmu.2018.00838 C5928262!5928262!29740443     free     free     free Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\29740443.jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       Front+Immunol 2018 ; 9 (ä): ä Nephropedia Template TP {{tp|p=29740443|t=2018. Endothelial Activation: The Ang/Tie Axis in Sepsis |pdf=|usr=}}{{29740443}} gab.com Text Endothelial Activation: The Ang/Tie Axis in Sepsis JO: Front Immunol http://www.kidney.de/pget.php?&tw=1&pmid=29740443 #FOAvip Sepsis, a dysregulated host response to infection that causes life-threatening organ dysfunction, is a highly heterogeneous syndrome with no specific treatment. Although sepsis can be caused by a wide variety of pathogenic organisms, endothelial dysfunction leading to vascular leak is a common mechanism of injury that contributes to the morbidity and mortality associated with the syndrome. Perturbations to the angiopoietin (Ang)/Tie2 axis cause endothelial cell activation and contribute to the pathogenesis of sepsis. In this review, we summarize how the Ang/Tie2 pathway is implicated in sepsis and describe its prognostic as well as therapeutic utility in life-threatening infections. Twit Text FOAVip Endothelial Activation: The Ang/Tie Axis in Sepsis ????Front Immunol http://www.kidney.de/pget.php?&tw=1&pmid=29740443 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=29740443 #FOAvip English Wikipedia <ref>{{Cite pmid|29740443}}</ref> Endothelial Activation: The Ang/Tie Axis in Sepsis #MMPMID29740443 Leligdowicz A; Richard-Greenblatt M; Wright J; Crowley VM; Kain KC Front Immunol 2018[]; 9 (ä): ä PMID29740443show ga Sepsis, a dysregulated host response to infection that causes life-threatening organ dysfunction, is a highly heterogeneous syndrome with no specific treatment. Although sepsis can be caused by a wide variety of pathogenic organisms, endothelial dysfunction leading to vascular leak is a common mechanism of injury that contributes to the morbidity and mortality associated with the syndrome. Perturbations to the angiopoietin (Ang)/Tie2 axis cause endothelial cell activation and contribute to the pathogenesis of sepsis. In this review, we summarize how the Ang/Tie2 pathway is implicated in sepsis and describe its prognostic as well as therapeutic utility in life-threatening infections. äEndothelial Activation: The Ang/Tie Axis in Sepsis (epmc).pdf DeepDyve Pubget Overpricing