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10.1186/s13054-018-2040-y http://scihub22266oqcxt.onion/10.1186/s13054-018-2040-y C5934860!5934860!29724256     free     free     free Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\29724256.jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       Crit+Care 2018 ; 22 (ä): ä Nephropedia Template TP {{tp|p=29724256|t=2018. Gut?kidney crosstalk in septic acute kidney injury |pdf=|usr=}}{{29724256}} gab.com Text Gut kidney crosstalk in septic acute kidney injury JO: Crit Care http://www.kidney.de/pget.php?&tw=1&pmid=29724256 #FOAvip Sepsis is the leading cause of acute kidney injury (AKI) in the intensive care unit (ICU). Septic AKI is a complex and multifactorial process that is incompletely understood. During sepsis, the disruption of the mucus membrane barrier, a shift in intestinal microbial flora, and microbial translocation may lead to systemic inflammation, which further alters host immune and metabolic homeostasis. This altered homeostasis may promote and potentiate the development of AKI. As part of this vicious cycle, when AKI develops, the clearance of inflammatory mediators and metabolic products is decreased. This will lead to further gut injury and breakdown in mucous membrane barriers. Thus, changes in the gut during sepsis can initiate and propagate septic AKI. This deleterious gut?kidney crosstalk may be a potential target for therapeutic maneuvers. This review analyses the underlying mechanisms in gut?kidney crosstalk in septic AKI. Twit Text FOAVip Gut kidney crosstalk in septic acute kidney injury ????Crit Care http://www.kidney.de/pget.php?&tw=1&pmid=29724256 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=29724256 #FOAvip English Wikipedia <ref>{{Cite pmid|29724256}}</ref> Gut?kidney crosstalk in septic acute kidney injury #MMPMID29724256 Zhang J; Ankawi G; Sun J; Digvijay K; Yin Y; Rosner MH; Ronco C Crit Care 2018[]; 22 (ä): ä PMID29724256show ga Sepsis is the leading cause of acute kidney injury (AKI) in the intensive care unit (ICU). Septic AKI is a complex and multifactorial process that is incompletely understood. During sepsis, the disruption of the mucus membrane barrier, a shift in intestinal microbial flora, and microbial translocation may lead to systemic inflammation, which further alters host immune and metabolic homeostasis. This altered homeostasis may promote and potentiate the development of AKI. As part of this vicious cycle, when AKI develops, the clearance of inflammatory mediators and metabolic products is decreased. This will lead to further gut injury and breakdown in mucous membrane barriers. Thus, changes in the gut during sepsis can initiate and propagate septic AKI. This deleterious gut?kidney crosstalk may be a potential target for therapeutic maneuvers. This review analyses the underlying mechanisms in gut?kidney crosstalk in septic AKI. äGut?kidney crosstalk in septic acute kidney injury (epmc).pdf DeepDyve Pubget Overpricing