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10.18632/oncotarget.11624 http://scihub22266oqcxt.onion/10.18632/oncotarget.11624 C5323236!5323236!27579619     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=27579619&cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215 Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\27579619.jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       Oncotarget 2016 ; 7 (40): 66310-22 Nephropedia Template TP {{tp|p=27579619|t=2016. I?B?: an emerging player in cancer |pdf=|usr=}}{{27579619}} gab.com Text I B : an emerging player in cancer JO: Oncotarget http://www.kidney.de/pget.php?&tw=1&pmid=27579619 #FOAvip I?B?, an atypical member of the nuclear I?B family of proteins, is expressed at low levels in most resting cells, but is induced upon stimulation of Toll-like/IL-1 receptors through an IRAK1/IRAK4/NF?B-dependent pathway. Like its homolog Bcl3, I?B? can regulate the transcription of a set of inflamatory genes through its association with the p50 or p52 subunits of NF-?B. Long studied as a key component of the immune response, I?B? emerges as an important regulator of inflammation, cell proliferation and survival. As a result, growing evidence support the role of this transcription factor in the pathogenesis number of human hematological and solid malignancies. Twit Text FOAVip I B : an emerging player in cancer ????Oncotarget http://www.kidney.de/pget.php?&tw=1&pmid=27579619 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=27579619 #FOAvip English Wikipedia <ref>{{Cite pmid|27579619}}</ref> I?B?: an emerging player in cancer #MMPMID27579619 Willems M; Dubois N; Musumeci L; Bours V; Robe PA Oncotarget 2016[Oct]; 7 (40): 66310-22 PMID27579619show ga I?B?, an atypical member of the nuclear I?B family of proteins, is expressed at low levels in most resting cells, but is induced upon stimulation of Toll-like/IL-1 receptors through an IRAK1/IRAK4/NF?B-dependent pathway. Like its homolog Bcl3, I?B? can regulate the transcription of a set of inflamatory genes through its association with the p50 or p52 subunits of NF-?B. Long studied as a key component of the immune response, I?B? emerges as an important regulator of inflammation, cell proliferation and survival. As a result, growing evidence support the role of this transcription factor in the pathogenesis number of human hematological and solid malignancies. äI?B?: an emerging player in cancer (epmc).pdf DeepDyve Pubget Overpricing