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10.4137/JEN.S25475 http://scihub22266oqcxt.onion/10.4137/JEN.S25475 C4822725!4822725!27081317     free     free     free Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\27081317.jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       J+Exp+Neurosci 2015 ; 9 (Suppl 2): 81-91 Nephropedia Template TP {{tp|p=27081317|t=2015. Lysosomal Storage Diseases?Regulating Neurodegeneration |pdf=|usr=}}{{27081317}} gab.com Text Lysosomal Storage Diseases Regulating Neurodegeneration JO: J Exp Neurosci http://www.kidney.de/pget.php?&tw=1&pmid=27081317 #FOAvip Autophagy is a complex pathway regulated by numerous signaling events that recycles macromolecules and can be perturbed in lysosomal storage diseases (LSDs). The concept of LSDs, which are characterized by aberrant, excessive storage of cellular material in lysosomes, developed following the discovery of an enzyme deficiency as the cause of Pompe disease in 1963. Great strides have since been made in better understanding the biology of LSDs. Defective lysosomal storage typically occurs in many cell types, but the nervous system, including the central nervous system and peripheral nervous system, is particularly vulnerable to LSDs, being affected in two-thirds of LSDs. This review provides a summary of some of the better characterized LSDs and the pathways affected in these disorders. Twit Text FOAVip Lysosomal Storage Diseases Regulating Neurodegeneration ????J Exp Neurosci http://www.kidney.de/pget.php?&tw=1&pmid=27081317 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=27081317 #FOAvip English Wikipedia <ref>{{Cite pmid|27081317}}</ref> Lysosomal Storage Diseases?Regulating Neurodegeneration #MMPMID27081317 Onyenwoke RU; Brenman JE J Exp Neurosci 2015[]; 9 (Suppl 2): 81-91 PMID27081317show ga Autophagy is a complex pathway regulated by numerous signaling events that recycles macromolecules and can be perturbed in lysosomal storage diseases (LSDs). The concept of LSDs, which are characterized by aberrant, excessive storage of cellular material in lysosomes, developed following the discovery of an enzyme deficiency as the cause of Pompe disease in 1963. Great strides have since been made in better understanding the biology of LSDs. Defective lysosomal storage typically occurs in many cell types, but the nervous system, including the central nervous system and peripheral nervous system, is particularly vulnerable to LSDs, being affected in two-thirds of LSDs. This review provides a summary of some of the better characterized LSDs and the pathways affected in these disorders. äLysosomal Storage Diseases?Regulating Neurodegeneration (epmc).pdf DeepDyve Pubget Overpricing