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10.1016/j.cll.2013.03.017 http://scihub22266oqcxt.onion/10.1016/j.cll.2013.03.017 C3685419!3685419!23702113     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=23702113&cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215 Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\23702113.jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       Clin+Lab+Med 2013 ; 33 (2): 207-33 Nephropedia Template TP {{tp|p=23702113|t=2013. New Concepts in Diabetic Embryopathy |pdf=|usr=}}{{23702113}} gab.com Text New Concepts in Diabetic Embryopathy JO: Clin Lab Med http://www.kidney.de/pget.php?&tw=1&pmid=23702113 #FOAvip Diabetes mellitus is responsible for nearly 10% of fetal anomalies in diabetic pregnancies. Although aggressive perinatal care and glycemic control are available in developed countries, the birth defect rate in diabetic pregnancies remains much higher than that in the general population. Major cellular activities--i.e., proliferation and apoptosis--and intracellular metabolic conditions--i.e., nitrosative, oxidative, and endoplasmic reticulum stress--have been demonstrated to be associated with diabetic embryopathy using animal models. Translating advances made in animal studies into clinical applications in humans will require collaborative efforts across the basic research, preclinical, and clinical communities. Twit Text FOAVip New Concepts in Diabetic Embryopathy ????Clin Lab Med http://www.kidney.de/pget.php?&tw=1&pmid=23702113 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=23702113 #FOAvip English Wikipedia <ref>{{Cite pmid|23702113}}</ref> New Concepts in Diabetic Embryopathy #MMPMID23702113 Zhao Z; Reece EA Clin Lab Med 2013[Jun]; 33 (2): 207-33 PMID23702113show ga Diabetes mellitus is responsible for nearly 10% of fetal anomalies in diabetic pregnancies. Although aggressive perinatal care and glycemic control are available in developed countries, the birth defect rate in diabetic pregnancies remains much higher than that in the general population. Major cellular activities--i.e., proliferation and apoptosis--and intracellular metabolic conditions--i.e., nitrosative, oxidative, and endoplasmic reticulum stress--have been demonstrated to be associated with diabetic embryopathy using animal models. Translating advances made in animal studies into clinical applications in humans will require collaborative efforts across the basic research, preclinical, and clinical communities. äNew Concepts in Diabetic Embryopathy (epmc).pdf DeepDyve Pubget Overpricing