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10.1007/s11899-015-0255-4 http://scihub22266oqcxt.onion/10.1007/s11899-015-0255-4 C4447552!4447552!25921388     free     free     free Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\25921388.jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       Curr+Hematol+Malig+Rep 2015 ; 10 (2): 126-31 Nephropedia Template TP {{tp|p=25921388|t=2015. Targeting the Microenvironment in Acute Myeloid Leukemia |pdf=|usr=}}{{25921388}} gab.com Text Targeting the Microenvironment in Acute Myeloid Leukemia JO: Curr Hematol Malig Rep http://www.kidney.de/pget.php?&tw=1&pmid=25921388 #FOAvip The bone marrow microenvironment plays a critical role in the development, progression, and relapse of acute myeloid leukemia (AML). Similar to normal hematopoietic stem cells, AML blasts express receptors on their surface, allowing them to interact with specific components of the marrow microenvironment. These interactions contribute to both chemotherapy resistance and disease relapse. Preclinical studies and early phase clinical trials have demonstrated the potential for targeting the tumor-microenvironment interactions in AML. Agents currently under investigation include hypoxia-inducible agents and inhibitors of CXCR4 and adhesion molecules such as VLA-4 and E-selectin. Twit Text FOAVip Targeting the Microenvironment in Acute Myeloid Leukemia ????Curr Hematol Malig Rep http://www.kidney.de/pget.php?&tw=1&pmid=25921388 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=25921388 #FOAvip English Wikipedia <ref>{{Cite pmid|25921388}}</ref> Targeting the Microenvironment in Acute Myeloid Leukemia #MMPMID25921388 Rashidi A; Uy GL Curr Hematol Malig Rep 2015[Jun]; 10 (2): 126-31 PMID25921388show ga The bone marrow microenvironment plays a critical role in the development, progression, and relapse of acute myeloid leukemia (AML). Similar to normal hematopoietic stem cells, AML blasts express receptors on their surface, allowing them to interact with specific components of the marrow microenvironment. These interactions contribute to both chemotherapy resistance and disease relapse. Preclinical studies and early phase clinical trials have demonstrated the potential for targeting the tumor-microenvironment interactions in AML. Agents currently under investigation include hypoxia-inducible agents and inhibitors of CXCR4 and adhesion molecules such as VLA-4 and E-selectin. äTargeting the Microenvironment in Acute Myeloid Leukemia (epmc).pdf DeepDyve Pubget Overpricing