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10.1101/cshperspect.a009753 http://scihub22266oqcxt.onion/10.1101/cshperspect.a009753 C3753720!3753720!23788646     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=23788646&cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215 Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\23788646.jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       Cold+Spring+Harb+Perspect+Med 2013 ; 3 (9): ä Nephropedia Template TP {{tp|p=23788646|t=2013. The Cystic Fibrosis Intestine |pdf=|usr=}}{{23788646}} gab.com Text The Cystic Fibrosis Intestine JO: Cold Spring Harb Perspect Med http://www.kidney.de/pget.php?&tw=1&pmid=23788646 #FOAvip The clinical manifestations of cystic fibrosis (CF) result from dysfunction of the cystic fibrosis transmembrane regulator protein (CFTR). The majority of people with CF have a limited life span as a consequence of CFTR dysfunction in the respiratory tract. However, CFTR dysfunction in the gastrointestinal (GI) tract occurs earlier in ontogeny and is present in all patients, regardless of genotype. The same pathophysiologic triad of obstruction, infection, and inflammation that causes disease in the airways also causes disease in the intestines. This article describes the effects of CFTR dysfunction on the intestinal tissues and the intraluminal environment. Mouse models of CF have greatly advanced our understanding of the GI manifestations of CF, which can be directly applied to understanding CF disease in humans. Twit Text FOAVip The Cystic Fibrosis Intestine ????Cold Spring Harb Perspect Med http://www.kidney.de/pget.php?&tw=1&pmid=23788646 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=23788646 #FOAvip English Wikipedia <ref>{{Cite pmid|23788646}}</ref> The Cystic Fibrosis Intestine #MMPMID23788646 De Lisle RC; Borowitz D Cold Spring Harb Perspect Med 2013[Sep]; 3 (9): ä PMID23788646show ga The clinical manifestations of cystic fibrosis (CF) result from dysfunction of the cystic fibrosis transmembrane regulator protein (CFTR). The majority of people with CF have a limited life span as a consequence of CFTR dysfunction in the respiratory tract. However, CFTR dysfunction in the gastrointestinal (GI) tract occurs earlier in ontogeny and is present in all patients, regardless of genotype. The same pathophysiologic triad of obstruction, infection, and inflammation that causes disease in the airways also causes disease in the intestines. This article describes the effects of CFTR dysfunction on the intestinal tissues and the intraluminal environment. Mouse models of CF have greatly advanced our understanding of the GI manifestations of CF, which can be directly applied to understanding CF disease in humans. äThe Cystic Fibrosis Intestine (epmc).pdf DeepDyve Pubget Overpricing