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10.1007/s12020-015-0838-9 http://scihub22266oqcxt.onion/10.1007/s12020-015-0838-9 C4824806!4824806!26754661     free     free     free Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\26754661.jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       Endocrine 2016 ; 52 (ä): 187-93 Nephropedia Template TP {{tp|p=26754661|t=2016. The role of ANGPTL3 in controlling lipoprotein metabolism |pdf=|usr=}}{{26754661}} gab.com Text The role of ANGPTL3 in controlling lipoprotein metabolism JO: Endocrine http://www.kidney.de/pget.php?&tw=1&pmid=26754661 #FOAvip Angiopoietin-like protein 3 (ANGPTL3) is a secretory protein regulating plasma lipid levels via affecting lipoprotein lipase- and endothelial lipase-mediated hydrolysis of triglycerides and phospholipids. ANGPTL3-deficiency due to loss-of-function mutations in the ANGPTL3 gene causes familial combined hypobetalipoproteinemia (FHBL2, OMIM # 605019), a phenotype characterized by low concentration of all major lipoprotein classes in circulation. ANGPTL3 is therefore a potential therapeutic target to treat combined hyperlipidemia, a major risk factor for atherosclerotic coronary heart disease. This review focuses on the mechanisms behind ANGPTL3-deficiency induced FHBL2. Twit Text FOAVip The role of ANGPTL3 in controlling lipoprotein metabolism ????Endocrine http://www.kidney.de/pget.php?&tw=1&pmid=26754661 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=26754661 #FOAvip English Wikipedia <ref>{{Cite pmid|26754661}}</ref> The role of ANGPTL3 in controlling lipoprotein metabolism #MMPMID26754661 Tikka A; Jauhiainen M Endocrine 2016[]; 52 (ä): 187-93 PMID26754661show ga Angiopoietin-like protein 3 (ANGPTL3) is a secretory protein regulating plasma lipid levels via affecting lipoprotein lipase- and endothelial lipase-mediated hydrolysis of triglycerides and phospholipids. ANGPTL3-deficiency due to loss-of-function mutations in the ANGPTL3 gene causes familial combined hypobetalipoproteinemia (FHBL2, OMIM # 605019), a phenotype characterized by low concentration of all major lipoprotein classes in circulation. ANGPTL3 is therefore a potential therapeutic target to treat combined hyperlipidemia, a major risk factor for atherosclerotic coronary heart disease. This review focuses on the mechanisms behind ANGPTL3-deficiency induced FHBL2. äThe role of ANGPTL3 in controlling lipoprotein metabolism (epmc).pdf DeepDyve Pubget Overpricing