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10.1016/j.trecan.2017.03.003 http://scihub22266oqcxt.onion/10.1016/j.trecan.2017.03.003 C5518481!5518481!28718438     free     free     free Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\28718438.jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       Trends+Cancer 2017 ; 3 (4): 259-68 Nephropedia Template TP {{tp|p=28718438|t=2017. Tissue-Intrinsic Tumor Hotspots: Terroir for Tumorigenesis |pdf=|usr=}}{{28718438}} gab.com Text Tissue-Intrinsic Tumor Hotspots: Terroir for Tumorigenesis JO: Trends Cancer http://www.kidney.de/pget.php?&tw=1&pmid=28718438 #FOAvip Epithelial tissues are highly organized systems with a remarkable homeostatic ability to maintain morphology through regulation of cellular proliferation and tissue integrity. This robust self-organizing system is progressively disrupted during tumor development. Recent studies of conserved tumor-suppressor genes in Drosophila showed how pro-tumor cells deviate from the robustly organized tissue microenvironment to take the first steps into becoming aggressive tumors. Here we review the ?tumor hotspot? hypothesis that explains how the tissue-intrinsic local microenvironment has a pivotal role in the initial stage of tumorigenesis in Drosophila epithelia and discuss comparable mechanisms in mammalian tissues. Twit Text FOAVip Tissue-Intrinsic Tumor Hotspots: Terroir for Tumorigenesis ????Trends Cancer http://www.kidney.de/pget.php?&tw=1&pmid=28718438 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=28718438 #FOAvip English Wikipedia <ref>{{Cite pmid|28718438}}</ref> Tissue-Intrinsic Tumor Hotspots: Terroir for Tumorigenesis #MMPMID28718438 Tamori Y; Deng WM Trends Cancer 2017[Apr]; 3 (4): 259-68 PMID28718438show ga Epithelial tissues are highly organized systems with a remarkable homeostatic ability to maintain morphology through regulation of cellular proliferation and tissue integrity. This robust self-organizing system is progressively disrupted during tumor development. Recent studies of conserved tumor-suppressor genes in Drosophila showed how pro-tumor cells deviate from the robustly organized tissue microenvironment to take the first steps into becoming aggressive tumors. Here we review the ?tumor hotspot? hypothesis that explains how the tissue-intrinsic local microenvironment has a pivotal role in the initial stage of tumorigenesis in Drosophila epithelia and discuss comparable mechanisms in mammalian tissues. äTissue-Intrinsic Tumor Hotspots: Terroir for Tumorigenesis (epmc).pdf DeepDyve Pubget Overpricing