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http://scihub22266oqcxt.onion/ 10535323!ä!10535323 Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\10535323.jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       Int+J+Dev+Biol 1999 ; 43 (5): 453-6 Nephropedia Template TP {{tp|p=10535323|t=1999. Influence of fetal environment on kidney development |pdf=|usr=}}{{10535323}} gab.com Text Influence of fetal environment on kidney development JO: Int J Dev Biol http://www.kidney.de/pget.php?&tw=1&pmid=10535323 #FOAvip Several lines of evidence, mostly derived from animal studies, indicate that changes in fetal environment may affect renal development. Besides maternal hyperglycemia or drug exposure, that were recently found to alter nephrogenesis, changes in vitamin A supply to the fetus may prove to be responsible for most of the variations in nephron number found in the population. A low vitamin A status in the fetus may be a major cause of inborn nephron deficit, either as a feature of intrauterine growth retardation or independently of growth retardation. The possibility that vitamin A status may also influence renal vascular development is raised. We suggest that low vitamin A supply to the fetus plays a role in the intrauterine programming of chronic renal disease and hypertension. Twit Text FOAVip Influence of fetal environment on kidney development ????Int J Dev Biol http://www.kidney.de/pget.php?&tw=1&pmid=10535323 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=10535323 #FOAvip English Wikipedia <ref>{{Cite pmid|10535323}}</ref> Influence of fetal environment on kidney development #MMPMID10535323 Merlet-Benichou C Int J Dev Biol 1999[]; 43 (5): 453-6 PMID10535323show ga Several lines of evidence, mostly derived from animal studies, indicate that changes in fetal environment may affect renal development. Besides maternal hyperglycemia or drug exposure, that were recently found to alter nephrogenesis, changes in vitamin A supply to the fetus may prove to be responsible for most of the variations in nephron number found in the population. A low vitamin A status in the fetus may be a major cause of inborn nephron deficit, either as a feature of intrauterine growth retardation or independently of growth retardation. The possibility that vitamin A status may also influence renal vascular development is raised. We suggest that low vitamin A supply to the fetus plays a role in the intrauterine programming of chronic renal disease and hypertension. |Animals[MESH] |Embryonic and Fetal Development/drug effects[MESH] |Female[MESH] |Humans[MESH] |Hyperglycemia/metabolism/pathology[MESH] |Kidney Diseases/congenital/metabolism/*pathology[MESH] |Kidney/abnormalities/drug effects/*embryology[MESH] |Nephrons/abnormalities/drug effects/*embryology[MESH] |Pregnancy[MESH] |Pregnancy Complications/metabolism/pathology[MESH] |Prenatal Exposure Delayed Effects[MESH]Influence of fetal environment on kidney development (epmc).pdf DeepDyve Pubget Overpricing