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http://scihub22266oqcxt.onion/ 11502076!ä!11502076 Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=11502076&cmd=llinks): Failed to open stream: HTTP request failed! 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Breach of IL-12 monopoly in the initiation of type 1 immunity to intracellular infections: IL-12 is not required |pdf=|usr=}}{{11502076}} gab.com Text Breach of IL-12 monopoly in the initiation of type 1 immunity to intracellular infections: IL-12 is not required JO: Cell Mol Biol (Noisy-le-grand) http://www.kidney.de/pget.php?&tw=1&pmid=11502076 #FOAvip IL-12 is believed to play an important role in type 1 T-cell differentiation and type 1 cytokine IFN-gamma release by T- and NK-cells and macrophages in host defense against intracellular infections by bacteria, parasites, fungi and viruses. However, recent studies by us and others have provided unequivocal evidence that while IL- 12 is critically required for the development of type 1 immunity to the majority of intracellular bacterial, parasitic and fungal infections, it is not required for anti-viral type 1 immune responses. These findings have provoked our re-thinking about the role of IL-12 in type 1 immunity and the search for additional cytokines capable of initiating anti-viral type 1 immunity. We hypothesize that there exist multiple cytokines including IL-12 which play a redundant role in the initiation of type 1 immunity against viral infection. These cytokines are likely released from not only antigen-presenting macrophages/dendritic cells but many other cell types, which suits the mode of viral infection. The existence of multiple factors capable of driving type 1 immunity endows the host with additional safeguards to cope with prevalent viral foes. Twit Text FOAVip Breach of IL-12 monopoly in the initiation of type 1 immunity to intracellular infections: IL-12 is not required ????Cell Mol Biol (Noisy-le-grand) http://www.kidney.de/pget.php?&tw=1&pmid=11502076 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=11502076 #FOAvip English Wikipedia <ref>{{Cite pmid|11502076}}</ref> Breach of IL-12 monopoly in the initiation of type 1 immunity to intracellular infections: IL-12 is not required #MMPMID11502076 Xing Z Cell Mol Biol (Noisy-le-grand) 2001[Jun]; 47 (4): 689-94 PMID11502076show ga IL-12 is believed to play an important role in type 1 T-cell differentiation and type 1 cytokine IFN-gamma release by T- and NK-cells and macrophages in host defense against intracellular infections by bacteria, parasites, fungi and viruses. However, recent studies by us and others have provided unequivocal evidence that while IL- 12 is critically required for the development of type 1 immunity to the majority of intracellular bacterial, parasitic and fungal infections, it is not required for anti-viral type 1 immune responses. These findings have provoked our re-thinking about the role of IL-12 in type 1 immunity and the search for additional cytokines capable of initiating anti-viral type 1 immunity. We hypothesize that there exist multiple cytokines including IL-12 which play a redundant role in the initiation of type 1 immunity against viral infection. These cytokines are likely released from not only antigen-presenting macrophages/dendritic cells but many other cell types, which suits the mode of viral infection. The existence of multiple factors capable of driving type 1 immunity endows the host with additional safeguards to cope with prevalent viral foes. |Animals[MESH] |Bacterial Infections/immunology[MESH] |Humans[MESH] |Influenza, Human/immunology[MESH] |Interferon-gamma/biosynthesis[MESH] |Interleukin-12/*immunology[MESH] |Lymphocytic Choriomeningitis/immunology[MESH] |Mycoses/immunology[MESH] |Parasitic Diseases/immunology[MESH] |Th1 Cells/*immunology[MESH]Breach of IL-12 monopoly in the initiation of type 1 immunity to intra(epmc).pdf DeepDyve Pubget Overpricing