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http://scihub22266oqcxt.onion/ 14217261!1927767!14217261     free     free     free Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\14217261.jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       Can+Med+Assoc+J 1964 ; 91 (18): 964-70 Nephropedia Template TP {{tp|p=14217261|t=1964. RECENT ADVANCES IN HYPERTENSION |pdf=|usr=}}{{14217261}} gab.com Text RECENT ADVANCES IN HYPERTENSION JO: Can Med Assoc J http://www.kidney.de/pget.php?&tw=1&pmid=14217261 #FOAvip The possible relationship between the renal mechanism of volume control and blood pressure regulation is discussed. Expansion of the extracellular fluid (ECF) and plasma volumes was demonstrated following renal artery constriction in the rat; after about one month ECF volume returned to normal although hypertension persisted. Measurements of cardiac output in the unanesthetized rat by an implanted electromagnetic flowmeter showed an initial rise in cardiac output after renal artery constriction, returning to normal in 10 to 15 days. A homeostatic hypothesis for the production of renal hypertension is put forward in which changes in ECF volume, capacity vessel tone and myocardial contractility participate in the development of hypertension by elevating cardiac output. Autoregulation of peripheral flow then occurs and the consequent restoration of blood pressure at a renal pressure receptor results in return to normal of cardiac output by negative feedback. Thus in chronic hypertension the high peripheral resistance is maintained by autoregulation. Twit Text FOAVip RECENT ADVANCES IN HYPERTENSION ????Can Med Assoc J http://www.kidney.de/pget.php?&tw=1&pmid=14217261 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=14217261 #FOAvip English Wikipedia <ref>{{Cite pmid|14217261}}</ref> RECENT ADVANCES IN HYPERTENSION #MMPMID14217261 WILSON C Can Med Assoc J 1964[Oct]; 91 (18): 964-70 PMID14217261show ga The possible relationship between the renal mechanism of volume control and blood pressure regulation is discussed. Expansion of the extracellular fluid (ECF) and plasma volumes was demonstrated following renal artery constriction in the rat; after about one month ECF volume returned to normal although hypertension persisted. Measurements of cardiac output in the unanesthetized rat by an implanted electromagnetic flowmeter showed an initial rise in cardiac output after renal artery constriction, returning to normal in 10 to 15 days. A homeostatic hypothesis for the production of renal hypertension is put forward in which changes in ECF volume, capacity vessel tone and myocardial contractility participate in the development of hypertension by elevating cardiac output. Autoregulation of peripheral flow then occurs and the consequent restoration of blood pressure at a renal pressure receptor results in return to normal of cardiac output by negative feedback. Thus in chronic hypertension the high peripheral resistance is maintained by autoregulation. |*Aldosterone[MESH] |*Angiotensins[MESH] |*Blood Pressure[MESH] |*Cardiac Output[MESH] |*Hypertension[MESH] |*Hypertension, Renal[MESH] |*Kidney[MESH] |*Physiology[MESH] |*Renal Artery Obstruction[MESH] |*Renin[MESH] |*Research[MESH] |*Sodium[MESH] |*Vascular Resistance[MESH] |*Vasomotor System[MESH] |*Water-Electrolyte Balance[MESH]RECENT ADVANCES IN HYPERTENSION (epmc).pdf DeepDyve Pubget Overpricing