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10.1111/j.1524-6175.2006.05110.x http://scihub22266oqcxt.onion/10.1111/j.1524-6175.2006.05110.x 16470083!8109360!16470083     free     free     free Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\16470083.jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       J+Clin+Hypertens+(Greenwich) 2006 ; 8 (2): 133-9 Nephropedia Template TP {{tp|p=16470083|t=2006. Aldosterone--a hormone of cardiovascular adaptation and maladaptation |pdf=|usr=}}{{16470083}} gab.com Text Aldosterone--a hormone of cardiovascular adaptation and maladaptation JO: J Clin Hypertens (Greenwich) http://www.kidney.de/pget.php?&tw=1&pmid=16470083 #FOAvip Aldosterone stimulates reabsorption of sodium, sustaining blood volume and pressure in the face of salt deprivation or extracellular fluid depletion. The steroid also stimulates excretion of potassium, protecting extracellular fluid from excessive levels of that ion. These two actions are relatively rapid and clearly adaptive when appropriately initiated and terminated, but maladaptive when prolonged or excessive, causing hypertension and electrolyte imbalance. Aldosterone and other mineralocorticoids exert slower, direct effects on cells in the heart, kidneys, and vessels, leading to hypertrophy, fibrosis, and dysfunction contributing to degenerative cardiovascular diseases. The maladaptive actions of aldosterone are exacerbated by sodium chloride, angiotensin, endothelin, and certain growth factors. Damage can be minimized by antagonists of aldosterone receptors, inhibitors of the renin system, depletion of salt, and repletion of potassium and magnesium. Specific inhibitors of fibrosis and hypertrophy, and more effective inhibitors of the renin system should be useful in the future. Twit Text FOAVip Aldosterone--a hormone of cardiovascular adaptation and maladaptation ????J Clin Hypertens (Greenwich) http://www.kidney.de/pget.php?&tw=1&pmid=16470083 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=16470083 #FOAvip English Wikipedia <ref>{{Cite pmid|16470083}}</ref> Aldosterone--a hormone of cardiovascular adaptation and maladaptation #MMPMID16470083 Goodfriend TL J Clin Hypertens (Greenwich) 2006[Feb]; 8 (2): 133-9 PMID16470083show ga Aldosterone stimulates reabsorption of sodium, sustaining blood volume and pressure in the face of salt deprivation or extracellular fluid depletion. The steroid also stimulates excretion of potassium, protecting extracellular fluid from excessive levels of that ion. These two actions are relatively rapid and clearly adaptive when appropriately initiated and terminated, but maladaptive when prolonged or excessive, causing hypertension and electrolyte imbalance. Aldosterone and other mineralocorticoids exert slower, direct effects on cells in the heart, kidneys, and vessels, leading to hypertrophy, fibrosis, and dysfunction contributing to degenerative cardiovascular diseases. The maladaptive actions of aldosterone are exacerbated by sodium chloride, angiotensin, endothelin, and certain growth factors. Damage can be minimized by antagonists of aldosterone receptors, inhibitors of the renin system, depletion of salt, and repletion of potassium and magnesium. Specific inhibitors of fibrosis and hypertrophy, and more effective inhibitors of the renin system should be useful in the future. |Aldosterone/*physiology[MESH] |Blood Pressure/*physiology[MESH] |Cardiovascular Diseases/etiology/*physiopathology[MESH] |Electrolytes/*metabolism[MESH] |Homeostasis[MESH] |Humans[MESH] |Hypertension/*physiopathology[MESH] |Potassium/metabolism[MESH] |Sodium/metabolism[MESH]Aldosterone--a hormone of cardiovascular adaptation and maladaptation (epmc).pdf DeepDyve Pubget Overpricing