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http://scihub22266oqcxt.onion/ 17348483!ä!17348483 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\17348483.jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       Isr+Med+Assoc+J 2007 ; 9 (2): 107-11 Nephropedia Template TP {{tp|p=17348483|t=2007. Renal slit diaphragm--the open zipper and the failing heart |pdf=|usr=}}{{17348483}} gab.com Text Renal slit diaphragm--the open zipper and the failing heart JO: Isr Med Assoc J http://www.kidney.de/pget.php?&tw=1&pmid=17348483 #FOAvip Inherited forms of proteinuria constitute a rare and heterogeneous group of diseases, the most prominent of which is glomerular dysfunction, which leads to proteinuria. Investigation of the genetic background underlying these diseases has provided significant data on the normal operation of the glomerular filter. Among the different components of the glomerulus, the podocyte slit diaphragm is considered the main source for genetically derived protein alteration, which leads in turn to proteinuria. Investigation of the different proteins revealed that the lack of nephrin and podocin is the leading cause of several inherited forms of proteinuria. It was also proposed that the lack of podocin is linked to cardiac anomalies. This review suggests that the absence of slit diaphragm proteins and the open zipper phenomenon are associated with cardiac anomalies. Twit Text FOAVip Renal slit diaphragm--the open zipper and the failing heart ????Isr Med Assoc J http://www.kidney.de/pget.php?&tw=1&pmid=17348483 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=17348483 #FOAvip English Wikipedia <ref>{{Cite pmid|17348483}}</ref> Renal slit diaphragm--the open zipper and the failing heart #MMPMID17348483 Blum S; Nakhoul F; Khankin E; Abassi Z Isr Med Assoc J 2007[Feb]; 9 (2): 107-11 PMID17348483show ga Inherited forms of proteinuria constitute a rare and heterogeneous group of diseases, the most prominent of which is glomerular dysfunction, which leads to proteinuria. Investigation of the genetic background underlying these diseases has provided significant data on the normal operation of the glomerular filter. Among the different components of the glomerulus, the podocyte slit diaphragm is considered the main source for genetically derived protein alteration, which leads in turn to proteinuria. Investigation of the different proteins revealed that the lack of nephrin and podocin is the leading cause of several inherited forms of proteinuria. It was also proposed that the lack of podocin is linked to cardiac anomalies. This review suggests that the absence of slit diaphragm proteins and the open zipper phenomenon are associated with cardiac anomalies. |Heart Failure/physiopathology[MESH] |Humans[MESH] |Intracellular Signaling Peptides and Proteins/*physiology[MESH] |Membrane Proteins/*physiology[MESH] |Nephrotic Syndrome/diagnosis/genetics/*pathology[MESH] |Podocytes[MESH]Renal slit diaphragm--the open zipper and the failing heart (epmc).pdf DeepDyve Pubget Overpricing