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10.1136/adc.2006.096958 http://scihub22266oqcxt.onion/10.1136/adc.2006.096958 17634184!2066071!17634184     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=17634184&cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215 Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\17634184.jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       Arch+Dis+Child 2007 ; 92 (12): 1136-40 Nephropedia Template TP {{tp|p=17634184|t=2007. Primary ciliary dyskinesia: current state of the art |pdf=|usr=}}{{17634184}} gab.com Text Primary ciliary dyskinesia: current state of the art JO: Arch Dis Child http://www.kidney.de/pget.php?&tw=1&pmid=17634184 #FOAvip Primary ciliary dyskinesia (PCD) is usually inherited as an autosomal recessive disorder and presents with upper and lower respiratory tract infection, and mirror image arrangement in around 50% of cases. Cilia dysfunction is also implicated in a wider spectrum of disease, including polycystic liver and kidney disease, central nervous system problems including retinopathy and hydrocephalus, and biliary atresia. Cilia are complex structures, containing more than 250 proteins; recent studies have begun to locate PCD genes scattered throughout the genome. Screening tests for PCD include nasal nitric oxide and in vivo tests of ciliary motility such as the saccharin test. Specific diagnosis requires examination of cilia by light and electron microscopy, with epithelial culture in doubtful cases. This is only available in supra-regional centres, recently centrally funded by the National Commissioning Group. Treatment is not evidence based and recommendations are largely extrapolated from cystic fibrosis and other suppurative lung diseases. Twit Text FOAVip Primary ciliary dyskinesia: current state of the art ????Arch Dis Child http://www.kidney.de/pget.php?&tw=1&pmid=17634184 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=17634184 #FOAvip English Wikipedia <ref>{{Cite pmid|17634184}}</ref> Primary ciliary dyskinesia: current state of the art #MMPMID17634184 Bush A; Chodhari R; Collins N; Copeland F; Hall P; Harcourt J; Hariri M; Hogg C; Lucas J; Mitchison HM; O'Callaghan C; Phillips G Arch Dis Child 2007[Dec]; 92 (12): 1136-40 PMID17634184show ga Primary ciliary dyskinesia (PCD) is usually inherited as an autosomal recessive disorder and presents with upper and lower respiratory tract infection, and mirror image arrangement in around 50% of cases. Cilia dysfunction is also implicated in a wider spectrum of disease, including polycystic liver and kidney disease, central nervous system problems including retinopathy and hydrocephalus, and biliary atresia. Cilia are complex structures, containing more than 250 proteins; recent studies have begun to locate PCD genes scattered throughout the genome. Screening tests for PCD include nasal nitric oxide and in vivo tests of ciliary motility such as the saccharin test. Specific diagnosis requires examination of cilia by light and electron microscopy, with epithelial culture in doubtful cases. This is only available in supra-regional centres, recently centrally funded by the National Commissioning Group. Treatment is not evidence based and recommendations are largely extrapolated from cystic fibrosis and other suppurative lung diseases. |Adolescent[MESH] |Adult[MESH] |Child[MESH] |Child, Preschool[MESH] |Cilia/physiology[MESH] |Humans[MESH] |Infant[MESH] |Infant, Newborn[MESH] |Kartagener Syndrome/complications/*diagnosis/genetics/*therapy[MESH] |Opportunistic Infections/complications/therapy[MESH]Primary ciliary dyskinesia: current state of the art (epmc).pdf DeepDyve Pubget Overpricing