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10.1007/s00424-008-0593-3 http://scihub22266oqcxt.onion/10.1007/s00424-008-0593-3 18839206!2730119!18839206     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=18839206&cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\18839206.jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       Pflugers+Arch 2009 ; 458 (1): 157-68 Nephropedia Template TP {{tp|p=18839206|t=2009. Regulation of potassium (K) handling in the renal collecting duct |pdf=|usr=}}{{18839206}} gab.com Text Regulation of potassium (K) handling in the renal collecting duct JO: Pflugers Arch http://www.kidney.de/pget.php?&tw=1&pmid=18839206 #FOAvip This review provides an overview of the molecular mechanisms of K transport in the mammalian connecting tubule (CNT) and cortical collecting duct (CCD), both nephron segments responsible for the regulation of renal K secretion. Aldosterone and dietary K intake are two of the most important factors regulating K secretion in the CNT and CCD. Recently, angiotensin II (AngII) has also been shown to play a role in the regulation of K secretion. In addition, genetic and molecular biological approaches have further identified new mechanisms by which aldosterone and dietary K intake regulate K transport. Thus, the interaction between serum-glucocorticoid-induced kinase 1 (SGK1) and with-no-lysine kinase 4 (WNK4) plays a significant role in mediating the effect of aldosterone on ROMK (Kir1.1), an important apical K channel modulating K secretion. Recent evidence suggests that WNK1, mitogen-activated protein kinases such as P38, ERK, and Src family protein tyrosine kinase are involved in mediating the effect of low K intake on apical K secretory channels. Twit Text FOAVip Regulation of potassium (K) handling in the renal collecting duct ????Pflugers Arch http://www.kidney.de/pget.php?&tw=1&pmid=18839206 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=18839206 #FOAvip English Wikipedia <ref>{{Cite pmid|18839206}}</ref> Regulation of potassium (K) handling in the renal collecting duct #MMPMID18839206 Wang WH; Giebisch G Pflugers Arch 2009[May]; 458 (1): 157-68 PMID18839206show ga This review provides an overview of the molecular mechanisms of K transport in the mammalian connecting tubule (CNT) and cortical collecting duct (CCD), both nephron segments responsible for the regulation of renal K secretion. Aldosterone and dietary K intake are two of the most important factors regulating K secretion in the CNT and CCD. Recently, angiotensin II (AngII) has also been shown to play a role in the regulation of K secretion. In addition, genetic and molecular biological approaches have further identified new mechanisms by which aldosterone and dietary K intake regulate K transport. Thus, the interaction between serum-glucocorticoid-induced kinase 1 (SGK1) and with-no-lysine kinase 4 (WNK4) plays a significant role in mediating the effect of aldosterone on ROMK (Kir1.1), an important apical K channel modulating K secretion. Recent evidence suggests that WNK1, mitogen-activated protein kinases such as P38, ERK, and Src family protein tyrosine kinase are involved in mediating the effect of low K intake on apical K secretory channels. |Aldosterone/physiology[MESH] |Angiotensin II/physiology[MESH] |Animals[MESH] |Extracellular Signal-Regulated MAP Kinases/physiology[MESH] |Gastrointestinal Hormones/physiology[MESH] |Immediate-Early Proteins/metabolism[MESH] |Kidney Tubules, Collecting/drug effects/*metabolism[MESH] |Natriuretic Peptides/physiology[MESH] |Potassium Channels, Inwardly Rectifying/physiology[MESH] |Potassium, Dietary/*pharmacology[MESH] |Potassium/*metabolism[MESH] |Protein Serine-Threonine Kinases/metabolism/physiology[MESH] |p38 Mitogen-Activated Protein Kinases/physiology[MESH]Regulation of potassium (K) handling in the renal collecting duct (epmc).pdf DeepDyve Pubget Overpricing