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10.1016/j.jcv.2008.10.008 http://scihub22266oqcxt.onion/10.1016/j.jcv.2008.10.008 19058999!ä!19058999 Deprecated: Implicit conversion from float 219.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 219.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 219.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\19058999.jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       J+Clin+Virol 2009 ; 44 (1): 33-8 Nephropedia Template TP {{tp|p=19058999|t=2009. Placental endothelial cells can be productively infected by Parvovirus B19 |pdf=|usr=}}{{19058999}} gab.com Text Placental endothelial cells can be productively infected by Parvovirus B19 JO: J Clin Virol http://www.kidney.de/pget.php?&tw=1&pmid=19058999 #FOAvip BACKGROUND: Parvovirus B19 vertical transmission occurs in about 30% of cases of maternal infection and may result in foetal hydrops or intrauterine foetal death. Details on the mechanism of transplacental transmission of B19 virus and subsequent foetal infection have not been elucidated. OBJECTIVE: To investigate the extent and distribution of B19 virus infection in placental tissues. STUDY DESIGN: Virological, histological, electron microscopy, immunohistochemical and immunofluorescence analysis of placental tissues obtained from a case of intrauterine foetal death caused by B19 virus. RESULTS: Real-time PCR analysis showed B19 virus DNA in placental samples. Histology, immunohistochemistry and electron microscopy demonstrated the concomitant infection of both foetal erythroid precursors and placental endothelial cells. In situ hybridisation for B19 virus nucleic acids, immunohistochemistry for B19 virus proteins and double labelling immunofluorescence confirmed that endothelial cells were productively infected by B19 virus. CONCLUSION: Foetal capillary endothelium in placental villi can be an additional target of productive B19 virus infection. Infection of placental endothelial cells may lead to a structural and functional damage critical both for altering maternal-foetal blood exchanges and for spreading the infection to the foetus, possibly concurring to the development of foetal hydrops and intrauterine foetal death. Twit Text FOAVip Placental endothelial cells can be productively infected by Parvovirus B19 ????J Clin Virol http://www.kidney.de/pget.php?&tw=1&pmid=19058999 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=19058999 #FOAvip English Wikipedia <ref>{{Cite pmid|19058999}}</ref> Placental endothelial cells can be productively infected by Parvovirus B19 #MMPMID19058999 Pasquinelli G; Bonvicini F; Foroni L; Salfi N; Gallinella G J Clin Virol 2009[Jan]; 44 (1): 33-8 PMID19058999show ga BACKGROUND: Parvovirus B19 vertical transmission occurs in about 30% of cases of maternal infection and may result in foetal hydrops or intrauterine foetal death. Details on the mechanism of transplacental transmission of B19 virus and subsequent foetal infection have not been elucidated. OBJECTIVE: To investigate the extent and distribution of B19 virus infection in placental tissues. STUDY DESIGN: Virological, histological, electron microscopy, immunohistochemical and immunofluorescence analysis of placental tissues obtained from a case of intrauterine foetal death caused by B19 virus. RESULTS: Real-time PCR analysis showed B19 virus DNA in placental samples. Histology, immunohistochemistry and electron microscopy demonstrated the concomitant infection of both foetal erythroid precursors and placental endothelial cells. In situ hybridisation for B19 virus nucleic acids, immunohistochemistry for B19 virus proteins and double labelling immunofluorescence confirmed that endothelial cells were productively infected by B19 virus. CONCLUSION: Foetal capillary endothelium in placental villi can be an additional target of productive B19 virus infection. Infection of placental endothelial cells may lead to a structural and functional damage critical both for altering maternal-foetal blood exchanges and for spreading the infection to the foetus, possibly concurring to the development of foetal hydrops and intrauterine foetal death. |Adult[MESH] |Animals[MESH] |Cells, Cultured[MESH] |DNA, Viral/isolation & purification[MESH] |Endothelial Cells/ultrastructure/*virology[MESH] |Erythroid Precursor Cells/virology[MESH] |Female[MESH] |Humans[MESH] |Parvoviridae Infections/*pathology/*transmission[MESH] |Parvovirus B19, Human/*growth & development[MESH] |Placenta/pathology/*virology[MESH] |Polymerase Chain Reaction/methods[MESH]Placental endothelial cells can be productively infected by Parvovirus(epmc).pdf DeepDyve Pubget Overpricing