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http://scihub22266oqcxt.onion/ 192954!ä!192954 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\192954.jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       Mayo+Clin+Proc 1977 ; 52 (5): 291-307 Nephropedia Template TP {{tp|p=192954|t=1977. The role of the adrenal cortex in human essential hypertension: keynote address |pdf=|usr=}}{{192954}} gab.com Text The role of the adrenal cortex in human essential hypertension: keynote address JO: Mayo Clin Proc http://www.kidney.de/pget.php?&tw=1&pmid=192954 #FOAvip The data presented establish that in early or mild essential hypertension there is a state of inappropriate hypermineralocorticoid activity represented by the sum of aldosterone and 18-hydroxy-11-deoxycorticosterone concentrations in the plasma. This disturbance, associated with a "normal" or excessive salt intake, would produce the arteriolar cationic changes in sodium, potassium, calcium, or magnesium leading to hypersensitivity or hyperresponsiveness of the arteriolar actomyosin to normal levels of circulating norepinephrine or angiotensin. The nature of the cationic changes in the arteriolar cells responsible for the increased tonicity of the arteriolar actomyosin, which is the fundamental cause of essential hypertension, remains to be elucidated. Twit Text FOAVip The role of the adrenal cortex in human essential hypertension: keynote address ????Mayo Clin Proc http://www.kidney.de/pget.php?&tw=1&pmid=192954 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=192954 #FOAvip English Wikipedia <ref>{{Cite pmid|192954}}</ref> The role of the adrenal cortex in human essential hypertension: keynote address #MMPMID192954 Genest J; Nowaczynski W; Kuchel O; Boucher R; Rojo-Ortega JM Mayo Clin Proc 1977[May]; 52 (5): 291-307 PMID192954show ga The data presented establish that in early or mild essential hypertension there is a state of inappropriate hypermineralocorticoid activity represented by the sum of aldosterone and 18-hydroxy-11-deoxycorticosterone concentrations in the plasma. This disturbance, associated with a "normal" or excessive salt intake, would produce the arteriolar cationic changes in sodium, potassium, calcium, or magnesium leading to hypersensitivity or hyperresponsiveness of the arteriolar actomyosin to normal levels of circulating norepinephrine or angiotensin. The nature of the cationic changes in the arteriolar cells responsible for the increased tonicity of the arteriolar actomyosin, which is the fundamental cause of essential hypertension, remains to be elucidated. |18-Hydroxydesoxycorticosterone/blood[MESH] |Adolescent[MESH] |Adrenal Cortex/*physiopathology[MESH] |Adrenal Glands/*physiopathology[MESH] |Adrenocorticotropic Hormone/pharmacology[MESH] |Adult[MESH] |Age Factors[MESH] |Aged[MESH] |Aldosterone/blood[MESH] |Angiotensin II/pharmacology[MESH] |Arteries/metabolism[MESH] |Blood Pressure[MESH] |Blood Volume[MESH] |Circadian Rhythm[MESH] |Female[MESH] |Humans[MESH] |Hypertension/*physiopathology[MESH] |Ions/metabolism[MESH] |Liver/blood supply[MESH] |Male[MESH] |Metabolic Clearance Rate[MESH] |Middle Aged[MESH] |Mineralocorticoids/metabolism[MESH] |Models, Biological[MESH] |Posture[MESH] |Radioimmunoassay[MESH] |Regional Blood Flow[MESH]The role of the adrenal cortex in human essential hypertension: keynot(epmc).pdf DeepDyve Pubget Overpricing