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10.1684/mrh.2009.0189 http://scihub22266oqcxt.onion/10.1684/mrh.2009.0189 20228000!ä!20228000 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 245.2 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 245.2 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 245.2 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 245.2 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\20228000.jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       Magnes+Res 2009 ; 22 (4): 225-34 Nephropedia Template TP {{tp|p=20228000|t=2009. Are the transient receptor potential melastatin (TRPM) channels important in magnesium homeostasis following traumatic brain injury?|pdf=|usr=}}{{20228000}} gab.com Text Are the transient receptor potential melastatin (TRPM) channels important in magnesium homeostasis following traumatic brain injury JO: Magnes Res http://www.kidney.de/pget.php?&tw=1&pmid=20228000 #FOAvip Traumatic brain injury (TBI) confers a major burden to Western society and effective treatments are urgently required to improve the long-term deficits that inflict TBI survivors. Depletion of intracellular Mg(2+) is a well-known phenomenon occurring after TBI and is associated with poor neurological outcome. However, despite success in pre-clinical experimental studies, therapies utilizing Mg(2+) have not always proven to be clinically effective. Recent evidence implicates members of the transient receptor potential melastatin (TRPM) channel family in processes leading to neuronal cell death following ischemic injury, however, the exact mechanism by which this occurs is not completely understood. Specifically, TRPM7 and TRPM6 are two channels that have been identified as potentially playing a role in regulating Mg(2+) homeostasis, although whether this role in magnesium regulation and neuronal injury is significant is controversial. The purpose of this review is to explore the relationship between TRPM family members and Mg(2+) homeostasis, including their potential involvement in secondary injury processes leading to cell death following TBI. Twit Text FOAVip Are the transient receptor potential melastatin (TRPM) channels important in magnesium homeostasis following traumatic brain injury ????Magnes Res http://www.kidney.de/pget.php?&tw=1&pmid=20228000 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=20228000 #FOAvip English Wikipedia <ref>{{Cite pmid|20228000}}</ref> Are the transient receptor potential melastatin (TRPM) channels important in magnesium homeostasis following traumatic brain injury? #MMPMID20228000 Cook NL; Van Den Heuvel C; Vink R Magnes Res 2009[Dec]; 22 (4): 225-34 PMID20228000show ga Traumatic brain injury (TBI) confers a major burden to Western society and effective treatments are urgently required to improve the long-term deficits that inflict TBI survivors. Depletion of intracellular Mg(2+) is a well-known phenomenon occurring after TBI and is associated with poor neurological outcome. However, despite success in pre-clinical experimental studies, therapies utilizing Mg(2+) have not always proven to be clinically effective. Recent evidence implicates members of the transient receptor potential melastatin (TRPM) channel family in processes leading to neuronal cell death following ischemic injury, however, the exact mechanism by which this occurs is not completely understood. Specifically, TRPM7 and TRPM6 are two channels that have been identified as potentially playing a role in regulating Mg(2+) homeostasis, although whether this role in magnesium regulation and neuronal injury is significant is controversial. The purpose of this review is to explore the relationship between TRPM family members and Mg(2+) homeostasis, including their potential involvement in secondary injury processes leading to cell death following TBI. |Brain Injuries/mortality/pathology/*physiopathology/therapy[MESH] |Calcium/physiology[MESH] |Cell Death[MESH] |Homeostasis[MESH] |Humans[MESH] |Magnesium/*physiology[MESH] |Receptors, N-Methyl-D-Aspartate/physiology[MESH] |Survivors[MESH]Are the transient receptor potential melastatin (TRPM) channels import(epmc).pdf DeepDyve Pubget Overpricing