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10.1681/ASN.2009070759 http://scihub22266oqcxt.onion/10.1681/ASN.2009070759 20360309!4577295!20360309     free     free     free Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\20360309.jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       J+Am+Soc+Nephrol 2010 ; 21 (7): 1093-6 Nephropedia Template TP {{tp|p=20360309|t=2010. Macula densa sensing and signaling mechanisms of renin release |pdf=|usr=}}{{20360309}} gab.com Text Macula densa sensing and signaling mechanisms of renin release JO: J Am Soc Nephrol http://www.kidney.de/pget.php?&tw=1&pmid=20360309 #FOAvip Macula densa cells in the distal nephron, according to the classic paradigm, are salt sensors that generate paracrine chemical signals in the juxtaglomerular apparatus to control vital kidney functions, including renal blood flow, glomerular filtration, and renin release. Renin is the rate-limiting step in the activation of the renin-angiotensin system, a key modulator of body fluid homeostasis. Here, we discuss recent advances in understanding macula densa sensing and suggest these cells, in addition to salt, also sense various chemical and metabolic signals in the tubular environment that directly trigger renin release. Twit Text FOAVip Macula densa sensing and signaling mechanisms of renin release ????J Am Soc Nephrol http://www.kidney.de/pget.php?&tw=1&pmid=20360309 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=20360309 #FOAvip English Wikipedia <ref>{{Cite pmid|20360309}}</ref> Macula densa sensing and signaling mechanisms of renin release #MMPMID20360309 Peti-Peterdi J; Harris RC J Am Soc Nephrol 2010[Jul]; 21 (7): 1093-6 PMID20360309show ga Macula densa cells in the distal nephron, according to the classic paradigm, are salt sensors that generate paracrine chemical signals in the juxtaglomerular apparatus to control vital kidney functions, including renal blood flow, glomerular filtration, and renin release. Renin is the rate-limiting step in the activation of the renin-angiotensin system, a key modulator of body fluid homeostasis. Here, we discuss recent advances in understanding macula densa sensing and suggest these cells, in addition to salt, also sense various chemical and metabolic signals in the tubular environment that directly trigger renin release. |Animals[MESH] |Glomerular Filtration Rate/physiology[MESH] |Homeostasis/physiology[MESH] |Humans[MESH] |Juxtaglomerular Apparatus/blood supply/*physiology[MESH] |Kidney Tubules, Distal/blood supply/*physiology[MESH] |Mice[MESH] |Mice, Knockout[MESH] |Models, Animal[MESH] |Nephrons/blood supply/*physiology[MESH] |Regional Blood Flow/physiology[MESH] |Renin-Angiotensin System/physiology[MESH] |Renin/*metabolism[MESH]Macula densa sensing and signaling mechanisms of renin release (epmc).pdf DeepDyve Pubget Overpricing