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10.1016/j.biocel.2010.05.014 http://scihub22266oqcxt.onion/10.1016/j.biocel.2010.05.014 20542138!2910191!20542138     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=20542138&cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\20542138.jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       Int+J+Biochem+Cell+Biol 2010 ; 42 (9): 1380-7 Nephropedia Template TP {{tp|p=20542138|t=2010. The glomerulus--a view from the outside--the podocyte |pdf=|usr=}}{{20542138}} gab.com Text The glomerulus--a view from the outside--the podocyte JO: Int J Biochem Cell Biol http://www.kidney.de/pget.php?&tw=1&pmid=20542138 #FOAvip In the past decade, podocyte research has been greatly aided by the development of powerful new molecular, cellular and animal tools, leading to elucidation of an increasing number of proteins involved in podocyte function and identification of mutated genes in hereditary glomerulopathies. Accumulating evidence indicates that podocyte disorders may not only underlie these hereditary glomerulopathies but also play crucial role in a broad spectrum of acquired glomerular diseases. Genetic susceptibility, environmental influence and systemic responses are all involved in the mediation of the pathogenesis of podocytopathies. Injured podocytes may predisopose to further injury of other podocytes and other adjacent/distant renal cells in a vicious cycle, leading to inexorable progression of glomerular injury. The classic view is that podocytes have a limited ability to proliferate in the normal mature kidney. However, recent research in rodents has provided suggestive evidence for podocyte regeneration resulting from differentiation of progenitor cells within Bowman's capsule. Twit Text FOAVip The glomerulus--a view from the outside--the podocyte ????Int J Biochem Cell Biol http://www.kidney.de/pget.php?&tw=1&pmid=20542138 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=20542138 #FOAvip English Wikipedia <ref>{{Cite pmid|20542138}}</ref> The glomerulus--a view from the outside--the podocyte #MMPMID20542138 Cheng H; Harris RC Int J Biochem Cell Biol 2010[Sep]; 42 (9): 1380-7 PMID20542138show ga In the past decade, podocyte research has been greatly aided by the development of powerful new molecular, cellular and animal tools, leading to elucidation of an increasing number of proteins involved in podocyte function and identification of mutated genes in hereditary glomerulopathies. Accumulating evidence indicates that podocyte disorders may not only underlie these hereditary glomerulopathies but also play crucial role in a broad spectrum of acquired glomerular diseases. Genetic susceptibility, environmental influence and systemic responses are all involved in the mediation of the pathogenesis of podocytopathies. Injured podocytes may predisopose to further injury of other podocytes and other adjacent/distant renal cells in a vicious cycle, leading to inexorable progression of glomerular injury. The classic view is that podocytes have a limited ability to proliferate in the normal mature kidney. However, recent research in rodents has provided suggestive evidence for podocyte regeneration resulting from differentiation of progenitor cells within Bowman's capsule. |Animals[MESH] |Bowman Capsule/cytology[MESH] |Cell Differentiation/physiology[MESH] |Humans[MESH] |Kidney Glomerulus/*cytology/*metabolism/pathology[MESH] |Kidney/cytology/metabolism/pathology[MESH] |Podocytes/*cytology/*metabolism/pathology[MESH]The glomerulus--a view from the outside--the podocyte (epmc).pdf DeepDyve Pubget Overpricing