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10.3265/Nefrologia.pre2010.Nov.10643 http://scihub22266oqcxt.onion/10.3265/Nefrologia.pre2010.Nov.10643 21407275!ä!21407275 Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=21407275&cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215 Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\21407275.jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       Nefrologia 2011 ; 31 (3): 268-74 Nephropedia Template TP {{tp|p=21407275|t=2011. Vascular and metabolic properties of manidipine |pdf=|usr=}}{{21407275}} gab.com Text Vascular and metabolic properties of manidipine JO: Nefrologia http://www.kidney.de/pget.php?&tw=1&pmid=21407275 #FOAvip The combination of renin-angiotensin system blockers with calcium channel blockers appears to be one of the most effective options for treating hypertension and diabetes.Nevertheless, not all calcium blockers behave in the same manner. Manidipine, unlike other third-generation dihydropyridine derived drugs, blocks T-type calcium channels present in the efferent glomerular arterioles, reducing intraglomerular pressure and microalbuminuria. In addition,T-type channels are related to proliferation, inflammation,fibrosis, vasoconstriction and activation of the renin-angiotensin system. The inhibition of these factors could explain the non-haemodynamic effects of manidipine as compared to other blockers. Twit Text FOAVip Vascular and metabolic properties of manidipine ????Nefrologia http://www.kidney.de/pget.php?&tw=1&pmid=21407275 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=21407275 #FOAvip English Wikipedia <ref>{{Cite pmid|21407275}}</ref> Vascular and metabolic properties of manidipine #MMPMID21407275 Buset Rios N; Rodriguez Esparragon F; Fernandez-Andrade Rodriguez C; Rodriguez Perez JC Nefrologia 2011[]; 31 (3): 268-74 PMID21407275show ga The combination of renin-angiotensin system blockers with calcium channel blockers appears to be one of the most effective options for treating hypertension and diabetes.Nevertheless, not all calcium blockers behave in the same manner. Manidipine, unlike other third-generation dihydropyridine derived drugs, blocks T-type calcium channels present in the efferent glomerular arterioles, reducing intraglomerular pressure and microalbuminuria. In addition,T-type channels are related to proliferation, inflammation,fibrosis, vasoconstriction and activation of the renin-angiotensin system. The inhibition of these factors could explain the non-haemodynamic effects of manidipine as compared to other blockers. |*Insulin Resistance[MESH] |Albuminuria/*drug therapy[MESH] |Calcium Channel Blockers/*metabolism/*pharmacology[MESH] |Cardiovascular Diseases[MESH] |Dihydropyridines/*pharmacology[MESH] |Humans[MESH] |Nitrobenzenes[MESH] |Oxidative Stress/*drug effects[MESH]Vascular and metabolic properties of manidipine (epmc).pdf DeepDyve Pubget Overpricing