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10.1155/2012/471823 http://scihub22266oqcxt.onion/10.1155/2012/471823 22474422!3296202!22474422     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=22474422&cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\22474422.jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       Exp+Diabetes+Res 2012 ; 2012 (ä): 471823 Nephropedia Template TP {{tp|p=22474422|t=2012. Strategies to reverse endothelial progenitor cell dysfunction in diabetes |pdf=|usr=}}{{22474422}} gab.com Text Strategies to reverse endothelial progenitor cell dysfunction in diabetes JO: Exp Diabetes Res http://www.kidney.de/pget.php?&tw=1&pmid=22474422 #FOAvip Bone-marrow-derived cells-mediated postnatal vasculogenesis has been reported as the main responsible for the regulation of vascular homeostasis in adults. Since their discovery, endothelial progenitor cells have been depicted as mediators of postnatal vasculogenesis for their peculiar phenotype (partially staminal and partially endothelial), their ability to differentiate in endothelial cell line and to be incorporated into the vessels wall during ischemia/damage. Diabetes mellitus, a condition characterized by cardiovascular disease, nephropathy, and micro- and macroangiopathy, showed a dysfunction of endothelial progenitor cells. Herein, we review the mechanisms involved in diabetes-related dysfunction of endothelial progenitor cells, highlighting how hyperglycemia affects the different steps of endothelial progenitor cells lifetime (i.e., bone marrow mobilization, trafficking into the bloodstream, differentiation in endothelial cells, and homing in damaged tissues/organs). Finally, we review preclinical and clinical strategies that aim to revert diabetes-induced dysfunction of endothelial progenitor cells as a means of finding new strategies to prevent diabetic complications. Twit Text FOAVip Strategies to reverse endothelial progenitor cell dysfunction in diabetes ????Exp Diabetes Res http://www.kidney.de/pget.php?&tw=1&pmid=22474422 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=22474422 #FOAvip English Wikipedia <ref>{{Cite pmid|22474422}}</ref> Strategies to reverse endothelial progenitor cell dysfunction in diabetes #MMPMID22474422 Petrelli A; Di Fenza R; Carvello M; Gatti F; Secchi A; Fiorina P Exp Diabetes Res 2012[]; 2012 (ä): 471823 PMID22474422show ga Bone-marrow-derived cells-mediated postnatal vasculogenesis has been reported as the main responsible for the regulation of vascular homeostasis in adults. Since their discovery, endothelial progenitor cells have been depicted as mediators of postnatal vasculogenesis for their peculiar phenotype (partially staminal and partially endothelial), their ability to differentiate in endothelial cell line and to be incorporated into the vessels wall during ischemia/damage. Diabetes mellitus, a condition characterized by cardiovascular disease, nephropathy, and micro- and macroangiopathy, showed a dysfunction of endothelial progenitor cells. Herein, we review the mechanisms involved in diabetes-related dysfunction of endothelial progenitor cells, highlighting how hyperglycemia affects the different steps of endothelial progenitor cells lifetime (i.e., bone marrow mobilization, trafficking into the bloodstream, differentiation in endothelial cells, and homing in damaged tissues/organs). Finally, we review preclinical and clinical strategies that aim to revert diabetes-induced dysfunction of endothelial progenitor cells as a means of finding new strategies to prevent diabetic complications. |Cell Differentiation/physiology[MESH] |Diabetes Mellitus/*physiopathology[MESH] |Endothelial Cells/cytology/*physiology[MESH] |Humans[MESH]Strategies to reverse endothelial progenitor cell dysfunction in diabe(epmc).pdf DeepDyve Pubget Overpricing