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10.4161/auto.23691 http://scihub22266oqcxt.onion/10.4161/auto.23691 23388380!3627663!23388380     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=23388380&cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\23388380.jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       Autophagy 2013 ; 9 (4): 451-8 Nephropedia Template TP {{tp|p=23388380|t=2013. DAMPs and autophagy: cellular adaptation to injury and unscheduled cell death |pdf=|usr=}}{{23388380}} gab.com Text DAMPs and autophagy: cellular adaptation to injury and unscheduled cell death JO: Autophagy http://www.kidney.de/pget.php?&tw=1&pmid=23388380 #FOAvip Autophagy is a lysosome-mediated catabolic process involving the degradation of intracellular contents (e.g., proteins and organelles) as well as invading microbes (e.g., parasites, bacteria and viruses). Multiple forms of cellular stress can stimulate this pathway, including nutritional imbalances, oxygen deprivation, immunological response, genetic defects, chromosomal anomalies and cytotoxic stress. Damage-associated molecular pattern molecules (DAMPs) are released by stressed cells undergoing autophagy or injury, and act as endogenous danger signals to regulate the subsequent inflammatory and immune response. A complex relationship exists between DAMPs and autophagy in cellular adaption to injury and unscheduled cell death. Since both autophagy and DAMPs are important for pathogenesis of human disease, it is crucial to understand how they interplay to sustain homeostasis in stressful or dangerous environments. Twit Text FOAVip DAMPs and autophagy: cellular adaptation to injury and unscheduled cell death ????Autophagy http://www.kidney.de/pget.php?&tw=1&pmid=23388380 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=23388380 #FOAvip English Wikipedia <ref>{{Cite pmid|23388380}}</ref> DAMPs and autophagy: cellular adaptation to injury and unscheduled cell death #MMPMID23388380 Zhang Q; Kang R; Zeh HJ 3rd; Lotze MT; Tang D Autophagy 2013[Apr]; 9 (4): 451-8 PMID23388380show ga Autophagy is a lysosome-mediated catabolic process involving the degradation of intracellular contents (e.g., proteins and organelles) as well as invading microbes (e.g., parasites, bacteria and viruses). Multiple forms of cellular stress can stimulate this pathway, including nutritional imbalances, oxygen deprivation, immunological response, genetic defects, chromosomal anomalies and cytotoxic stress. Damage-associated molecular pattern molecules (DAMPs) are released by stressed cells undergoing autophagy or injury, and act as endogenous danger signals to regulate the subsequent inflammatory and immune response. A complex relationship exists between DAMPs and autophagy in cellular adaption to injury and unscheduled cell death. Since both autophagy and DAMPs are important for pathogenesis of human disease, it is crucial to understand how they interplay to sustain homeostasis in stressful or dangerous environments. |*Adaptation, Physiological[MESH] |*Autophagy[MESH] |*Stress, Physiological[MESH] |Animals[MESH] |Humans[MESH] |Lysosomes/metabolism[MESH] |Models, Biological[MESH]DAMPs and autophagy: cellular adaptation to injury and unscheduled cel(epmc).pdf DeepDyve Pubget Overpricing