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10.1007/s40139-017-0139-5 http://scihub22266oqcxt.onion/10.1007/s40139-017-0139-5 29038744!5640172!29038744     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=29038744&cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215 Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\29038744.jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       Curr+Pathobiol+Rep 2017 ; 5 (2): 153-159 Nephropedia Template TP {{tp|p=29038744|t=2017. Autophagy and Ferroptosis - What s the Connection?|pdf=|usr=}}{{29038744}} gab.com Text Autophagy and Ferroptosis - What s the Connection JO: Curr Pathobiol Rep http://www.kidney.de/pget.php?&tw=1&pmid=29038744 #FOAvip PURPOSE OF REVIEW: Autophagy is a conserved intracellular degradation system and plays a dual role in cell death, depending on context and phase. Ferroptosis is a new form of regulated cell death that mainly depends on iron accumulation and lipid peroxidation. In this review, we summarize the processes of autophagy and ferroptosis and discuss their crosstalk mechanisms at the molecular level. RECENT FINDINGS: The original study shows that ferroptosis is morphologically, biochemically, and genetically distinct from autophagy and other types of cell death. However, recent studies demonstrate that activation of ferroptosis is indeed dependent on the induction of autophagy. Additionally, many ferroptosis regulators such as SLC7A11, GPX4, NRF2, p53, HSPB1, CISD1, FANCD2, and ACSL4 have been identified as potential regulators of autophagy. SUMMARY: This review not only highlights the importance of autophagy as an emerging mechanism of ferroptosis, but also raises new insights regarding regulated cell death. Twit Text FOAVip Autophagy and Ferroptosis - What s the Connection ????Curr Pathobiol Rep http://www.kidney.de/pget.php?&tw=1&pmid=29038744 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=29038744 #FOAvip English Wikipedia <ref>{{Cite pmid|29038744}}</ref> Autophagy and Ferroptosis - What s the Connection? #MMPMID29038744 Kang R; Tang D Curr Pathobiol Rep 2017[Jun]; 5 (2): 153-159 PMID29038744show ga PURPOSE OF REVIEW: Autophagy is a conserved intracellular degradation system and plays a dual role in cell death, depending on context and phase. Ferroptosis is a new form of regulated cell death that mainly depends on iron accumulation and lipid peroxidation. In this review, we summarize the processes of autophagy and ferroptosis and discuss their crosstalk mechanisms at the molecular level. RECENT FINDINGS: The original study shows that ferroptosis is morphologically, biochemically, and genetically distinct from autophagy and other types of cell death. However, recent studies demonstrate that activation of ferroptosis is indeed dependent on the induction of autophagy. Additionally, many ferroptosis regulators such as SLC7A11, GPX4, NRF2, p53, HSPB1, CISD1, FANCD2, and ACSL4 have been identified as potential regulators of autophagy. SUMMARY: This review not only highlights the importance of autophagy as an emerging mechanism of ferroptosis, but also raises new insights regarding regulated cell death. äAutophagy and Ferroptosis - What s the Connection?(epmc).pdf DeepDyve Pubget Overpricing