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10.1038/s41418-017-0012-4 http://scihub22266oqcxt.onion/10.1038/s41418-017-0012-4 29362479!5864239!29362479     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=29362479&cmd=llinks): Failed to open stream: HTTP request failed! 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Molecular mechanisms of cell death: recommendations of the Nomenclature Committee on Cell Death 2018 |pdf=|usr=}}{{29362479}} gab.com Text Molecular mechanisms of cell death: recommendations of the Nomenclature Committee on Cell Death 2018 JO: Cell Death Differ http://www.kidney.de/pget.php?&tw=1&pmid=29362479 #FOAvip Over the past decade, the Nomenclature Committee on Cell Death (NCCD) has formulated guidelines for the definition and interpretation of cell death from morphological, biochemical, and functional perspectives. Since the field continues to expand and novel mechanisms that orchestrate multiple cell death pathways are unveiled, we propose an updated classification of cell death subroutines focusing on mechanistic and essential (as opposed to correlative and dispensable) aspects of the process. As we provide molecularly oriented definitions of terms including intrinsic apoptosis, extrinsic apoptosis, mitochondrial permeability transition (MPT)-driven necrosis, necroptosis, ferroptosis, pyroptosis, parthanatos, entotic cell death, NETotic cell death, lysosome-dependent cell death, autophagy-dependent cell death, immunogenic cell death, cellular senescence, and mitotic catastrophe, we discuss the utility of neologisms that refer to highly specialized instances of these processes. The mission of the NCCD is to provide a widely accepted nomenclature on cell death in support of the continued development of the field. Twit Text FOAVip Molecular mechanisms of cell death: recommendations of the Nomenclature Committee on Cell Death 2018 ????Cell Death Differ http://www.kidney.de/pget.php?&tw=1&pmid=29362479 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=29362479 #FOAvip English Wikipedia <ref>{{Cite pmid|29362479}}</ref> Molecular mechanisms of cell death: recommendations of the Nomenclature Committee on Cell Death 2018 #MMPMID29362479 Galluzzi L; Vitale I; Aaronson SA; Abrams JM; Adam D; Agostinis P; Alnemri ES; Altucci L; Amelio I; Andrews DW; Annicchiarico-Petruzzelli M; Antonov AV; Arama E; Baehrecke EH; Barlev NA; Bazan NG; Bernassola F; Bertrand MJM; Bianchi K; Blagosklonny MV; Blomgren K; Borner C; Boya P; Brenner C; Campanella M; Candi E; Carmona-Gutierrez D; Cecconi F; Chan FK; Chandel NS; Cheng EH; Chipuk JE; Cidlowski JA; Ciechanover A; Cohen GM; Conrad M; Cubillos-Ruiz JR; Czabotar PE; D'Angiolella V; Dawson TM; Dawson VL; De Laurenzi V; De Maria R; Debatin KM; DeBerardinis RJ; Deshmukh M; Di Daniele N; Di Virgilio F; Dixit VM; Dixon SJ; Duckett CS; Dynlacht BD; El-Deiry WS; Elrod JW; Fimia GM; Fulda S; Garcia-Saez AJ; Garg AD; Garrido C; Gavathiotis E; Golstein P; Gottlieb E; Green DR; Greene LA; Gronemeyer H; Gross A; Hajnoczky G; Hardwick JM; Harris IS; Hengartner MO; Hetz C; Ichijo H; Jaattela M; Joseph B; Jost PJ; Juin PP; Kaiser WJ; Karin M; Kaufmann T; Kepp O; Kimchi A; Kitsis RN; Klionsky DJ; Knight RA; Kumar S; Lee SW; Lemasters JJ; Levine B; Linkermann A; Lipton SA; Lockshin RA; Lopez-Otin C; Lowe SW; Luedde T; Lugli E; MacFarlane M; Madeo F; Malewicz M; Malorni W; Manic G; Marine JC; Martin SJ; Martinou JC; Medema JP; Mehlen P; Meier P; Melino S; Miao EA; Molkentin JD; Moll UM; Munoz-Pinedo C; Nagata S; Nunez G; Oberst A; Oren M; Overholtzer M; Pagano M; Panaretakis T; Pasparakis M; Penninger JM; Pereira DM; Pervaiz S; Peter ME; Piacentini M; Pinton P; Prehn JHM; Puthalakath H; Rabinovich GA; Rehm M; Rizzuto R; Rodrigues CMP; Rubinsztein DC; Rudel T; Ryan KM; Sayan E; Scorrano L; Shao F; Shi Y; Silke J; Simon HU; Sistigu A; Stockwell BR; Strasser A; Szabadkai G; Tait SWG; Tang D; Tavernarakis N; Thorburn A; Tsujimoto Y; Turk B; Vanden Berghe T; Vandenabeele P; Vander Heiden MG; Villunger A; Virgin HW; Vousden KH; Vucic D; Wagner EF; Walczak H; Wallach D; Wang Y; Wells JA; Wood W; Yuan J; Zakeri Z; Zhivotovsky B; Zitvogel L; Melino G; Kroemer G Cell Death Differ 2018[Mar]; 25 (3): 486-541 PMID29362479show ga Over the past decade, the Nomenclature Committee on Cell Death (NCCD) has formulated guidelines for the definition and interpretation of cell death from morphological, biochemical, and functional perspectives. Since the field continues to expand and novel mechanisms that orchestrate multiple cell death pathways are unveiled, we propose an updated classification of cell death subroutines focusing on mechanistic and essential (as opposed to correlative and dispensable) aspects of the process. As we provide molecularly oriented definitions of terms including intrinsic apoptosis, extrinsic apoptosis, mitochondrial permeability transition (MPT)-driven necrosis, necroptosis, ferroptosis, pyroptosis, parthanatos, entotic cell death, NETotic cell death, lysosome-dependent cell death, autophagy-dependent cell death, immunogenic cell death, cellular senescence, and mitotic catastrophe, we discuss the utility of neologisms that refer to highly specialized instances of these processes. The mission of the NCCD is to provide a widely accepted nomenclature on cell death in support of the continued development of the field. |*Cell Death[MESH] |Animals[MESH] |Humans[MESH] |Lysosomes/metabolism/pathology[MESH] |Mitochondrial Membrane Transport Proteins/metabolism[MESH] |Mitochondrial Permeability Transition Pore[MESH]Molecular mechanisms of cell death: recommendations of the Nomenclatur(epmc).pdf DeepDyve Pubget Overpricing