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10.1016/j.cellimm.2020.104078 http://scihub22266oqcxt.onion/10.1016/j.cellimm.2020.104078 32164997!ä!32164997 Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=32164997&cmd=llinks): Failed to open stream: HTTP request failed! 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Pulmonary inflammatory response to influenza virus infection in pigs is regulated by DAP12 and macrophage M1 and M2 phenotypes |pdf=|usr=}}{{32164997}} gab.com Text Pulmonary inflammatory response to influenza virus infection in pigs is regulated by DAP12 and macrophage M1 and M2 phenotypes JO: Cell Immunol http://www.kidney.de/pget.php?&tw=1&pmid=32164997 #FOAvip We delineated the expression of DAP12 (DNAX-Activating Protein) and its associated receptors, TREM-1, TREM-2 and MDL-1 in pig alveolar monocyte/macrophages (AMM) that have attained M1 or M2 phenotypes. Pig AMM stimulated in vitro with IFN-gamma and IL-4 induced the expression of M1 (TNFalpha and iNOS) and M2 (ARG1 and no MMR) phenotypic markers, respectively. In influenza virus infected pigs at seven days post-infection, in addition to substantial modulations in the M1 and M2 markers expression, DAP12, TREM-1 and MDL-1 were downregulated in AMM. Thus, DAP12 signaling promoted the anti-inflammatory pathway in AMM of influenza virus infected pigs. Twit Text FOAVip Pulmonary inflammatory response to influenza virus infection in pigs is regulated by DAP12 and macrophage M1 and M2 phenotypes ????Cell Immunol http://www.kidney.de/pget.php?&tw=1&pmid=32164997 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=32164997 #FOAvip English Wikipedia <ref>{{Cite pmid|32164997}}</ref> Pulmonary inflammatory response to influenza virus infection in pigs is regulated by DAP12 and macrophage M1 and M2 phenotypes #MMPMID32164997 Hiremath J; Renu S; Tabynov K; Renukaradhya GJ Cell Immunol 2020[Jun]; 352 (ä): 104078 PMID32164997show ga We delineated the expression of DAP12 (DNAX-Activating Protein) and its associated receptors, TREM-1, TREM-2 and MDL-1 in pig alveolar monocyte/macrophages (AMM) that have attained M1 or M2 phenotypes. Pig AMM stimulated in vitro with IFN-gamma and IL-4 induced the expression of M1 (TNFalpha and iNOS) and M2 (ARG1 and no MMR) phenotypic markers, respectively. In influenza virus infected pigs at seven days post-infection, in addition to substantial modulations in the M1 and M2 markers expression, DAP12, TREM-1 and MDL-1 were downregulated in AMM. Thus, DAP12 signaling promoted the anti-inflammatory pathway in AMM of influenza virus infected pigs. |Adaptor Proteins, Signal Transducing/*immunology/metabolism[MESH] |Animals[MESH] |Biomarkers/metabolism[MESH] |Cytokines/metabolism[MESH] |Female[MESH] |Influenza A virus/immunology/pathogenicity[MESH] |Macrophages, Alveolar/*immunology/metabolism[MESH] |Macrophages/immunology/metabolism[MESH] |Male[MESH] |Membrane Proteins/*immunology/metabolism[MESH] |Monocytes/metabolism[MESH] |Orthomyxoviridae Infections/*immunology[MESH] |Orthomyxoviridae/metabolism[MESH] |Phenotype[MESH] |Signal Transduction[MESH] |Swine/immunology[MESH]Pulmonary inflammatory response to influenza virus infection in pigs i(epmc).pdf DeepDyve Pubget Overpricing