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10.1016/j.cca.2020.04.027 http://scihub22266oqcxt.onion/10.1016/j.cca.2020.04.027 32348783!7195008!32348783     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=32348783&cmd=llinks): Failed to open stream: HTTP request failed! 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Hyperinflammation and derangement of renin-angiotensin-aldosterone system in COVID-19: A novel hypothesis for clinically suspected hypercoagulopathy and microvascular immunothrombosis |pdf=|usr=}}{{32348783}} gab.com Text Hyperinflammation and derangement of renin-angiotensin-aldosterone system in COVID-19: A novel hypothesis for clinically suspected hypercoagulopathy and microvascular immunothrombosis JO: Clin Chim Acta http://www.kidney.de/pget.php?&tw=1&pmid=32348783 #FOAvip Early clinical evidence suggests that severe cases of coronavirus disease 2019 (COVID-19), caused by the severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2), are frequently characterized by hyperinflammation, imbalance of renin-angiotensin-aldosterone system, and a particular form of vasculopathy, thrombotic microangiopathy, and intravascular coagulopathy. In this paper, we present an immunothrombosis model of COVID-19. We discuss the underlying pathogenesis and the interaction between multiple systems, resulting in propagation of immunothrombosis, which through investigation in the coming weeks, may lead to both an improved understanding of COVID-19 pathophysiology and identification of innovative and efficient therapeutic targets to reverse the otherwise unfavorable clinical outcome of many of these patients. Twit Text FOAVip Hyperinflammation and derangement of renin-angiotensin-aldosterone system in COVID-19: A novel hypothesis for clinically suspected hypercoagulopathy and microvascular immunothrombosis ????Clin Chim Acta http://www.kidney.de/pget.php?&tw=1&pmid=32348783 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=32348783 #FOAvip English Wikipedia <ref>{{Cite pmid|32348783}}</ref> Hyperinflammation and derangement of renin-angiotensin-aldosterone system in COVID-19: A novel hypothesis for clinically suspected hypercoagulopathy and microvascular immunothrombosis #MMPMID32348783 Henry BM; Vikse J; Benoit S; Favaloro EJ; Lippi G Clin Chim Acta 2020[Aug]; 507 (ä): 167-173 PMID32348783show ga Early clinical evidence suggests that severe cases of coronavirus disease 2019 (COVID-19), caused by the severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2), are frequently characterized by hyperinflammation, imbalance of renin-angiotensin-aldosterone system, and a particular form of vasculopathy, thrombotic microangiopathy, and intravascular coagulopathy. In this paper, we present an immunothrombosis model of COVID-19. We discuss the underlying pathogenesis and the interaction between multiple systems, resulting in propagation of immunothrombosis, which through investigation in the coming weeks, may lead to both an improved understanding of COVID-19 pathophysiology and identification of innovative and efficient therapeutic targets to reverse the otherwise unfavorable clinical outcome of many of these patients. |Aldosterone/*immunology[MESH] |Betacoronavirus/*immunology[MESH] |COVID-19[MESH] |Coronavirus Infections/*immunology/physiopathology[MESH] |Humans[MESH] |Immunity, Innate/immunology[MESH] |Inflammation/immunology/physiopathology/virology[MESH] |Microvessels/*immunology/physiopathology/virology[MESH] |Pandemics[MESH] |Pneumonia, Viral/*immunology/physiopathology[MESH] |Renin-Angiotensin System/*immunology[MESH] |SARS-CoV-2[MESH] |Thrombophilia/*immunology/physiopathology/virology[MESH]Hyperinflammation and derangement of renin-angiotensin-aldosterone sys(epmc).pdf DeepDyve Pubget Overpricing