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10.1016/j.lfs.2020.118510 http://scihub22266oqcxt.onion/10.1016/j.lfs.2020.118510 32991879!7521881!32991879     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=32991879&cmd=llinks): Failed to open stream: HTTP request failed! 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Pharmacological agents to therapeutic treatment of cardiac injury caused by Covid-19 |pdf=|usr=}}{{32991879}} gab.com Text Pharmacological agents to therapeutic treatment of cardiac injury caused by Covid-19 JO: Life Sci http://www.kidney.de/pget.php?&tw=1&pmid=32991879 #FOAvip SARS-CoV-2 is responsible for the 2019 coronavirus disease (COVID-19), a global pandemic that began in March 2020 and is currently in progress. To date, COVID-19 has caused about 935,000 deaths in more than 200 countries. The respiratory system is most affected by injuries caused by COVID-19, but other organs may be involved, including the cardiovascular system. SARS-CoV-2 penetrates host cells through the angiotensin 2 conversion enzyme (ACE-2). ACE-2 is expressed not only in the lungs, but also in other organs, including the cardiovascular system. Several studies have found that a good percentage of patients with severe COVID-19 have cardiac lesions, including myocardial fibrosis, edema and pericarditis. Pathological remodeling of the extracellular matrix caused by viral infection leads to myocardial fibrotic lesions. These fibrotic scars can cause cardiac dysfunction, reducing the ejection fraction caused by the presence of stiffened myocardial matrix, or cardiac arrhythmias that cause an alteration in the electrical conduction system of the heart. These cardiac dysfunctions can cause death. It is therefore essential to identify cardiac involvement early in order to act with appropriate therapeutic treatments. In this review, we describe what is known about cardiac injury from COVID-19, highlighting effective pharmacological therapeutic solutions to combat cardiac injury, particularly cardiac fibrosis, caused by COVID-19. Twit Text FOAVip Pharmacological agents to therapeutic treatment of cardiac injury caused by Covid-19 ????Life Sci http://www.kidney.de/pget.php?&tw=1&pmid=32991879 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=32991879 #FOAvip English Wikipedia <ref>{{Cite pmid|32991879}}</ref> Pharmacological agents to therapeutic treatment of cardiac injury caused by Covid-19 #MMPMID32991879 Vitiello A; Ferrara F Life Sci 2020[Dec]; 262 (�): 118510 PMID32991879show ga SARS-CoV-2 is responsible for the 2019 coronavirus disease (COVID-19), a global pandemic that began in March 2020 and is currently in progress. To date, COVID-19 has caused about 935,000 deaths in more than 200 countries. The respiratory system is most affected by injuries caused by COVID-19, but other organs may be involved, including the cardiovascular system. SARS-CoV-2 penetrates host cells through the angiotensin 2 conversion enzyme (ACE-2). ACE-2 is expressed not only in the lungs, but also in other organs, including the cardiovascular system. Several studies have found that a good percentage of patients with severe COVID-19 have cardiac lesions, including myocardial fibrosis, edema and pericarditis. Pathological remodeling of the extracellular matrix caused by viral infection leads to myocardial fibrotic lesions. These fibrotic scars can cause cardiac dysfunction, reducing the ejection fraction caused by the presence of stiffened myocardial matrix, or cardiac arrhythmias that cause an alteration in the electrical conduction system of the heart. These cardiac dysfunctions can cause death. It is therefore essential to identify cardiac involvement early in order to act with appropriate therapeutic treatments. In this review, we describe what is known about cardiac injury from COVID-19, highlighting effective pharmacological therapeutic solutions to combat cardiac injury, particularly cardiac fibrosis, caused by COVID-19. |*COVID-19 Drug Treatment[MESH] |Angiotensin-Converting Enzyme 2/physiology[MESH] |COVID-19/*complications/physiopathology[MESH] |Cardiomyopathies/*drug therapy/*etiology[MESH] |Humans[MESH] |Pandemics[MESH]Pharmacological agents to therapeutic treatment of cardiac injury caus(epmc).pdf DeepDyve Pubget Overpricing