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10.1186/s12245-021-00348-7 http://scihub22266oqcxt.onion/10.1186/s12245-021-00348-7 33882828!8058752!33882828     free     free     free Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\33882828.jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       Int+J+Emerg+Med 2021 ; 14 (1): 23 Nephropedia Template TP {{tp|p=33882828|t=2021. The pathophysiology of "happy" hypoglycemia |pdf=|usr=}}{{33882828}} gab.com Text The pathophysiology of "happy" hypoglycemia JO: Int J Emerg Med http://www.kidney.de/pget.php?&tw=1&pmid=33882828 #FOAvip BACKGROUND: Hypoglycemia usually includes various neurological symptoms, which are the consequence of neuroglycopenia. When it is severe, it is associated with altered mental status, even coma. CASE PRESENTATION: We report the case of a patient with severe hypoglycemia, completely asymptomatic, due to the increase of lactate production in response to tissue hypoperfusion following a hemorrhagic shock. This illustrates that lactate can substitute glucose as an energy substrate for the brain. It is also a reminder that this metabolite, despite its bad reputation maintained by its role as a marker of severity in critical care patients, has a fundamental role in our metabolism. CONCLUSIONS: Following the example of the "happy hypoxemia" recently reported in the literature describing asymptomatic hypoxemia in COVID-19 patients, we describe a case of "happy hypoglycemia." Twit Text FOAVip The pathophysiology of "happy" hypoglycemia ????Int J Emerg Med http://www.kidney.de/pget.php?&tw=1&pmid=33882828 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=33882828 #FOAvip English Wikipedia <ref>{{Cite pmid|33882828}}</ref> The pathophysiology of "happy" hypoglycemia #MMPMID33882828 Loeb T; Ozguler A; Baer G; Baer M Int J Emerg Med 2021[Apr]; 14 (1): 23 PMID33882828show ga BACKGROUND: Hypoglycemia usually includes various neurological symptoms, which are the consequence of neuroglycopenia. When it is severe, it is associated with altered mental status, even coma. CASE PRESENTATION: We report the case of a patient with severe hypoglycemia, completely asymptomatic, due to the increase of lactate production in response to tissue hypoperfusion following a hemorrhagic shock. This illustrates that lactate can substitute glucose as an energy substrate for the brain. It is also a reminder that this metabolite, despite its bad reputation maintained by its role as a marker of severity in critical care patients, has a fundamental role in our metabolism. CONCLUSIONS: Following the example of the "happy hypoxemia" recently reported in the literature describing asymptomatic hypoxemia in COVID-19 patients, we describe a case of "happy hypoglycemia." äThe pathophysiology of "happy" hypoglycemia (epmc).pdf DeepDyve Pubget Overpricing