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10.1099/0022-1317-68-2-425 http://scihub22266oqcxt.onion/10.1099/0022-1317-68-2-425 3819696!ä!3819696 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\3819696.jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       J+Gen+Virol 1987 ; 68 ( Pt 2) (ä): 425-32 Nephropedia Template TP {{tp|p=3819696|t=1987. Protective mechanisms against pulmonary infection with influenza virus I Relative contribution of polymorphonuclear leukocytes and of alveolar macrophages to protection during the early phase of intranasal infection |pdf=|usr=}}{{3819696}} gab.com Text Protective mechanisms against pulmonary infection with influenza virus I Relative contribution of polymorphonuclear leukocytes and of alveolar macrophages to protection during the early phase of intranasal infection JO: J Gen Virol http://www.kidney.de/pget.php?&tw=1&pmid=3819696 #FOAvip The relative contribution of polymorphonuclear leukocytes and macrophages in the early protection against intranasal infection of mice with influenza virus was investigated. Virus multiplication in the lung in the early phase of infection with less than 1.5 X 10(3) plaque-forming units was enhanced by X-ray irradiation. The intranasal administration of carrageenan did not influence the titre of virus. However, when mice were infected with 1.5 X 10(4) plaque-forming units, the virus titre was elevated by intranasal administration of carrageenan as well as by X-ray irradiation, but not by intraperitoneal administration of carrageenan. The intranasal administration of carrageenan not only inhibited the phagocytic activity of alveolar macrophages but also enhanced susceptibility to the virus. On the other hand, polymorphonuclear leukocytes were capable of phagocytosing the virus in vitro and were non-permissive for virus infection. Neutralizing antibody and interferon were not detectable in the early stage of the infection. These results suggested that polymorphonuclear leukocytes (X-ray-sensitive, carrageenan-resistant) were the cells primarily responsible for early protection in influenza virus infection and that after infection with a high dose of the virus alveolar macrophages (X-ray-resistant, carrageenan-sensitive) also played a protective role in the early phase. Twit Text FOAVip Protective mechanisms against pulmonary infection with influenza virus I Relative contribution of polymorphonuclear leukocytes and of alveolar macrophages to protection during the early phase of intranasal infection ????J Gen Virol http://www.kidney.de/pget.php?&tw=1&pmid=3819696 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=3819696 #FOAvip English Wikipedia <ref>{{Cite pmid|3819696}}</ref> Protective mechanisms against pulmonary infection with influenza virus I Relative contribution of polymorphonuclear leukocytes and of alveolar macrophages to protection during the early phase of intranasal infection #MMPMID3819696 Fujisawa H; Tsuru S; Taniguchi M; Zinnaka Y; Nomoto K J Gen Virol 1987[Feb]; 68 ( Pt 2) (ä): 425-32 PMID3819696show ga The relative contribution of polymorphonuclear leukocytes and macrophages in the early protection against intranasal infection of mice with influenza virus was investigated. Virus multiplication in the lung in the early phase of infection with less than 1.5 X 10(3) plaque-forming units was enhanced by X-ray irradiation. The intranasal administration of carrageenan did not influence the titre of virus. However, when mice were infected with 1.5 X 10(4) plaque-forming units, the virus titre was elevated by intranasal administration of carrageenan as well as by X-ray irradiation, but not by intraperitoneal administration of carrageenan. The intranasal administration of carrageenan not only inhibited the phagocytic activity of alveolar macrophages but also enhanced susceptibility to the virus. On the other hand, polymorphonuclear leukocytes were capable of phagocytosing the virus in vitro and were non-permissive for virus infection. Neutralizing antibody and interferon were not detectable in the early stage of the infection. These results suggested that polymorphonuclear leukocytes (X-ray-sensitive, carrageenan-resistant) were the cells primarily responsible for early protection in influenza virus infection and that after infection with a high dose of the virus alveolar macrophages (X-ray-resistant, carrageenan-sensitive) also played a protective role in the early phase. |Administration, Intranasal[MESH] |Animals[MESH] |Carrageenan/pharmacology[MESH] |Gamma Rays[MESH] |Influenza A virus/isolation & purification[MESH] |Luminescent Measurements[MESH] |Lung/microbiology[MESH] |Macrophages/*physiology[MESH] |Mice[MESH] |Neutrophils/*physiology[MESH] |Orthomyxoviridae Infections/*immunology[MESH] |Phagocytosis/drug effects/radiation effects[MESH]Protective mechanisms against pulmonary infection with influenza virus(epmc).pdf DeepDyve Pubget Overpricing