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10.3109/10641968309081802 http://scihub22266oqcxt.onion/10.3109/10641968309081802 6136351!ä!6136351 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\6136351.jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       Clin+Exp+Hypertens+A 1983 ; 5 (5): 691-719 Nephropedia Template TP {{tp|p=6136351|t=1983. Erythrocyte membrane abnormalities in hypertension: a comparison between two animal models |pdf=|usr=}}{{6136351}} gab.com Text Erythrocyte membrane abnormalities in hypertension: a comparison between two animal models JO: Clin Exp Hypertens A http://www.kidney.de/pget.php?&tw=1&pmid=6136351 #FOAvip Calcium-membrane interactions have been studied in two animal models of hypertension using the erythrocyte membrane as a model system. The Okamoto-Aoki strain of spontaneously hypertensive rats (SHR) was the first examined, and the activities of the Ca++/Mg++-ATPases in the membrane of SHR erythrocytes were found to be consistently higher than those of the normotensive controls (WKY), while other membrane enzymes such as Na+/K+-ATPase and acetylcholinesterase were not detectably altered. Erythrocyte membranes of the SHR also have a higher passive permeability to calcium as well as other functional and compositional differences when compared to those of the WKY. These findings suggest that the membrane alterations in the SHR may be related to an increased passive permeability to calcium, and a possibly compensatory increase in Ca++/Mg++-ATPase activity in these animals. The second animal model examined was the deoxycorticosterone/salt-induced hypertension (DOCA) in uninephrectomized rats. In DOCA rats with comparable degree of blood pressure elevation, none of the erythrocyte membrane abnormalities observed in the SHR were present, suggesting that the latter alterations are probably genetically determined and not a consequence of elevated arterial pressure. Twit Text FOAVip Erythrocyte membrane abnormalities in hypertension: a comparison between two animal models ????Clin Exp Hypertens A http://www.kidney.de/pget.php?&tw=1&pmid=6136351 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=6136351 #FOAvip English Wikipedia <ref>{{Cite pmid|6136351}}</ref> Erythrocyte membrane abnormalities in hypertension: a comparison between two animal models #MMPMID6136351 Chan TC; Godin DV; Sutter MC Clin Exp Hypertens A 1983[]; 5 (5): 691-719 PMID6136351show ga Calcium-membrane interactions have been studied in two animal models of hypertension using the erythrocyte membrane as a model system. The Okamoto-Aoki strain of spontaneously hypertensive rats (SHR) was the first examined, and the activities of the Ca++/Mg++-ATPases in the membrane of SHR erythrocytes were found to be consistently higher than those of the normotensive controls (WKY), while other membrane enzymes such as Na+/K+-ATPase and acetylcholinesterase were not detectably altered. Erythrocyte membranes of the SHR also have a higher passive permeability to calcium as well as other functional and compositional differences when compared to those of the WKY. These findings suggest that the membrane alterations in the SHR may be related to an increased passive permeability to calcium, and a possibly compensatory increase in Ca++/Mg++-ATPase activity in these animals. The second animal model examined was the deoxycorticosterone/salt-induced hypertension (DOCA) in uninephrectomized rats. In DOCA rats with comparable degree of blood pressure elevation, none of the erythrocyte membrane abnormalities observed in the SHR were present, suggesting that the latter alterations are probably genetically determined and not a consequence of elevated arterial pressure. |*Disease Models, Animal[MESH] |*Rats[MESH] |Adenosine Triphosphatases/blood[MESH] |Animals[MESH] |Ca(2+) Mg(2+)-ATPase[MESH] |Calcium-Transporting ATPases/blood[MESH] |Calcium/metabolism[MESH] |Cell Membrane Permeability[MESH] |Desoxycorticosterone/adverse effects[MESH] |Erythrocyte Membrane/*metabolism[MESH] |Erythrocytes/*metabolism[MESH] |Hypertension/*blood/chemically induced/genetics[MESH] |Osmotic Fragility[MESH] |Rats, Inbred Strains[MESH] |Sodium Chloride/adverse effects[MESH]Erythrocyte membrane abnormalities in hypertension: a comparison betwe(epmc).pdf DeepDyve Pubget Overpricing