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10.1152/ajprenal.1980.238.5.F380 http://scihub22266oqcxt.onion/10.1152/ajprenal.1980.238.5.F380 6246813!ä!6246813 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\6246813.jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       Am+J+Physiol 1980 ; 238 (5): F380-6 Nephropedia Template TP {{tp|p=6246813|t=1980. Renal potassium adaptation: Na-K-ATPase activity along the nephron after chronic potassium loading |pdf=|usr=}}{{6246813}} gab.com Text Renal potassium adaptation: Na-K-ATPase activity along the nephron after chronic potassium loading JO: Am J Physiol http://www.kidney.de/pget.php?&tw=1&pmid=6246813 #FOAvip To identify nephron sites where renal potassium adaptation takes place, Na-K-ATPase was measured with a micromethod along the nephron of potassium-loaded mice. A possible role of aldosterone in this process was evaluated in K-loaded animals treated concurrently with pharmacologic doses of spironolactone. Animals fed a potassium-enriched diet for at least 2 wk excreted about 90% of ingested potassium in the urine, and fractional potassium clearance averaged 87 +/- 8%, compared with 13 +/- 2% in controls. Na-K-ATPase activity per millimeter tubule length increased by 225% in the cortical collecting tubule and by 177% in the medullary collecting tubule, but was not substantially affected in other nephron segments. Stimulation of Na-K-ATPase was identical in the cortical collecting tubule of K-loaded mice treated with spironolactone. These results indicate that chronic potassium loading in the mouse results in an adaptive increase potassium loading in the mouse results in an adaptive increase in Na-K-ATPase in the collecting tubule and suggest that this nephron segment is the major site of potassium adaptation in this species. This effect appears to be independent of aldosterone. Twit Text FOAVip Renal potassium adaptation: Na-K-ATPase activity along the nephron after chronic potassium loading ????Am J Physiol http://www.kidney.de/pget.php?&tw=1&pmid=6246813 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=6246813 #FOAvip English Wikipedia <ref>{{Cite pmid|6246813}}</ref> Renal potassium adaptation: Na-K-ATPase activity along the nephron after chronic potassium loading #MMPMID6246813 Doucet A; Katz AI Am J Physiol 1980[May]; 238 (5): F380-6 PMID6246813show ga To identify nephron sites where renal potassium adaptation takes place, Na-K-ATPase was measured with a micromethod along the nephron of potassium-loaded mice. A possible role of aldosterone in this process was evaluated in K-loaded animals treated concurrently with pharmacologic doses of spironolactone. Animals fed a potassium-enriched diet for at least 2 wk excreted about 90% of ingested potassium in the urine, and fractional potassium clearance averaged 87 +/- 8%, compared with 13 +/- 2% in controls. Na-K-ATPase activity per millimeter tubule length increased by 225% in the cortical collecting tubule and by 177% in the medullary collecting tubule, but was not substantially affected in other nephron segments. Stimulation of Na-K-ATPase was identical in the cortical collecting tubule of K-loaded mice treated with spironolactone. These results indicate that chronic potassium loading in the mouse results in an adaptive increase potassium loading in the mouse results in an adaptive increase in Na-K-ATPase in the collecting tubule and suggest that this nephron segment is the major site of potassium adaptation in this species. This effect appears to be independent of aldosterone. |Adenosine Triphosphatases/metabolism[MESH] |Animals[MESH] |Diet[MESH] |Glomerular Filtration Rate[MESH] |Kidney Tubules, Collecting/enzymology[MESH] |Kidney Tubules, Distal/enzymology[MESH] |Kidney Tubules, Proximal/enzymology[MESH] |Kidney/physiology[MESH] |Magnesium/pharmacology[MESH] |Male[MESH] |Mice[MESH] |Nephrons/*enzymology[MESH] |Potassium/*pharmacology[MESH]Renal potassium adaptation: Na-K-ATPase activity along the nephron aft(epmc).pdf DeepDyve Pubget Overpricing