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10.1099/0022-1317-64-8-1807 http://scihub22266oqcxt.onion/10.1099/0022-1317-64-8-1807 6875518!ä!6875518 Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=6875518&cmd=llinks): Failed to open stream: HTTP request failed! 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The similar interaction of ferret alveolar macrophages with influenza virus strains of differing virulence at normal and pyrexial temperatures |pdf=|usr=}}{{6875518}} gab.com Text The similar interaction of ferret alveolar macrophages with influenza virus strains of differing virulence at normal and pyrexial temperatures JO: J Gen Virol http://www.kidney.de/pget.php?&tw=1&pmid=6875518 #FOAvip The possibility that ferret lung macrophages may be one factor operating in vivo to prevent infection of susceptible alveolar cells (as demonstrated by organ cultures) by both virulent and attenuated strains of influenza virus has been investigated. Phagocytosis of four strains of influenza virus [A/PR/8/34 (H1N1) and clone 64d (attenuated for ferrets) and clones 64c and 7a (virulent for ferrets) of the recombinant virus A/PR/8/34-A/England/939/69 (H3N2)] by ferret alveolar macrophages in vitro showed that all strains, whether virulent or attenuated, attached equally well (72 to 93%). Recoveries of virus were similar (17 to 44%) whether phagocytosis occurred at the normal temperature of the ferret (39 degrees C) or at pyrexial temperatures induced during infection (40 degrees C for A/PR/8/34 and clone 64d; 41 degrees C for clones 7a and 64c). Thus, alveolar macrophages probably contribute to the lack of alveolar infection observed in vivo. Twit Text FOAVip The similar interaction of ferret alveolar macrophages with influenza virus strains of differing virulence at normal and pyrexial temperatures ????J Gen Virol http://www.kidney.de/pget.php?&tw=1&pmid=6875518 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=6875518 #FOAvip English Wikipedia <ref>{{Cite pmid|6875518}}</ref> The similar interaction of ferret alveolar macrophages with influenza virus strains of differing virulence at normal and pyrexial temperatures #MMPMID6875518 Bird RA; Sweet C; Husseini RH; Smith H J Gen Virol 1983[Aug]; 64 (Pt 8) (ä): 1807-10 PMID6875518show ga The possibility that ferret lung macrophages may be one factor operating in vivo to prevent infection of susceptible alveolar cells (as demonstrated by organ cultures) by both virulent and attenuated strains of influenza virus has been investigated. Phagocytosis of four strains of influenza virus [A/PR/8/34 (H1N1) and clone 64d (attenuated for ferrets) and clones 64c and 7a (virulent for ferrets) of the recombinant virus A/PR/8/34-A/England/939/69 (H3N2)] by ferret alveolar macrophages in vitro showed that all strains, whether virulent or attenuated, attached equally well (72 to 93%). Recoveries of virus were similar (17 to 44%) whether phagocytosis occurred at the normal temperature of the ferret (39 degrees C) or at pyrexial temperatures induced during infection (40 degrees C for A/PR/8/34 and clone 64d; 41 degrees C for clones 7a and 64c). Thus, alveolar macrophages probably contribute to the lack of alveolar infection observed in vivo. |*Phagocytosis[MESH] |Animals[MESH] |Ferrets[MESH] |Influenza A virus/immunology/*pathogenicity[MESH] |Macrophages/*immunology/microbiology[MESH] |Pulmonary Alveoli/cytology[MESH] |Temperature[MESH]The similar interaction of ferret alveolar macrophages with influenza (epmc).pdf DeepDyve Pubget Overpricing