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10.1006/abbi.1994.1048 http://scihub22266oqcxt.onion/10.1006/abbi.1994.1048 7509147!ä!7509147 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\7509147.jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       Arch+Biochem+Biophys 1994 ; 308 (2): 335-41 Nephropedia Template TP {{tp|p=7509147|t=1994. Characterization of magnesium efflux from Ehrlich ascites tumor cells |pdf=|usr=}}{{7509147}} gab.com Text Characterization of magnesium efflux from Ehrlich ascites tumor cells JO: Arch Biochem Biophys http://www.kidney.de/pget.php?&tw=1&pmid=7509147 #FOAvip Magnesium efflux from intact Ehrlich ascites tumor cells (EATC) has been characterized under 0-trans conditions. It is shown that a magnesium-extruding mechanism operates in these cells which brings about magnesium efflux up to 20% of the cell total content. EATC magnesium efflux is independent from extracellular Ca2+ and its apparent velocity is not affected by [Mg2+]o up to 160 microM. This extrusion, however, is strictly dependent on extracellular Na+ and it is inhibited by ouabain (1 mM), amiloride (1 mM), imipramine (0.5 mM) and quinidine (1 mM). It is not affected by HMA (5-N,N-hexamethylene) amiloride) (0.5 microM) and vanadate (0.1 mM). Bumetanide (0.5 mM) enhances magnesium extrusion in these cells. EATC magnesium extrusion can be significantly stimulated by db cAMP, forskolin, and IBX (3-isobutyl-1-methyl-xanthine). The intracellular magnesium distribution, studied also by means of the ionophore A23187, is regulated by energy metabolism and cell ATP content. Altogether our data demonstrate that EATC exhibit a magnesium extrusion sustained by Na+ gradient across the plasma membrane and carried out by a Na+/Mg2+ antiport. In these cells magnesium homoeostasis results, regulated efficiently by cell ATP and cAMP content. Twit Text FOAVip Characterization of magnesium efflux from Ehrlich ascites tumor cells ????Arch Biochem Biophys http://www.kidney.de/pget.php?&tw=1&pmid=7509147 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=7509147 #FOAvip English Wikipedia <ref>{{Cite pmid|7509147}}</ref> Characterization of magnesium efflux from Ehrlich ascites tumor cells #MMPMID7509147 Wolf FI; Di Francesco A; Cittadini A Arch Biochem Biophys 1994[Feb]; 308 (2): 335-41 PMID7509147show ga Magnesium efflux from intact Ehrlich ascites tumor cells (EATC) has been characterized under 0-trans conditions. It is shown that a magnesium-extruding mechanism operates in these cells which brings about magnesium efflux up to 20% of the cell total content. EATC magnesium efflux is independent from extracellular Ca2+ and its apparent velocity is not affected by [Mg2+]o up to 160 microM. This extrusion, however, is strictly dependent on extracellular Na+ and it is inhibited by ouabain (1 mM), amiloride (1 mM), imipramine (0.5 mM) and quinidine (1 mM). It is not affected by HMA (5-N,N-hexamethylene) amiloride) (0.5 microM) and vanadate (0.1 mM). Bumetanide (0.5 mM) enhances magnesium extrusion in these cells. EATC magnesium extrusion can be significantly stimulated by db cAMP, forskolin, and IBX (3-isobutyl-1-methyl-xanthine). The intracellular magnesium distribution, studied also by means of the ionophore A23187, is regulated by energy metabolism and cell ATP content. Altogether our data demonstrate that EATC exhibit a magnesium extrusion sustained by Na+ gradient across the plasma membrane and carried out by a Na+/Mg2+ antiport. In these cells magnesium homoeostasis results, regulated efficiently by cell ATP and cAMP content. |1-Methyl-3-isobutylxanthine/pharmacology[MESH] |Adenosine Triphosphate/metabolism[MESH] |Amiloride/analogs & derivatives/pharmacology[MESH] |Animals[MESH] |Antimycin A/pharmacology[MESH] |Bucladesine/pharmacology[MESH] |Bumetanide/pharmacology[MESH] |Calcimycin/pharmacology[MESH] |Carcinoma, Ehrlich Tumor/*metabolism[MESH] |Colforsin/pharmacology[MESH] |Cyclic AMP/metabolism[MESH] |Energy Metabolism[MESH] |Glucose/pharmacology[MESH] |Homeostasis[MESH] |Imipramine/pharmacology[MESH] |Kinetics[MESH] |Magnesium/*metabolism[MESH] |Mice[MESH] |Ouabain/pharmacology[MESH] |Quinidine/pharmacology[MESH] |Rotenone/pharmacology[MESH] |Time Factors[MESH]Characterization of magnesium efflux from Ehrlich ascites tumor cells (epmc).pdf DeepDyve Pubget Overpricing