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10.1161/STROKEAHA.109.558346 http://scihub22266oqcxt.onion/10.1161/STROKEAHA.109.558346 C2774207!2774207!19644062     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=19644062&cmd=llinks): Failed to open stream: HTTP request failed! 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Gene Variation of the Transient Receptor Potential Cation Channel, Subfamily M, Member 7 (TRPM7) and Risk of Incident Ischemic Stroke: A Prospective, Nested Case-Control Study |pdf=|usr=}}{{19644062}} gab.com Text Gene Variation of the Transient Receptor Potential Cation Channel, Subfamily M, Member 7 (TRPM7) and Risk of Incident Ischemic Stroke: A Prospective, Nested Case-Control Study JO: Stroke http://www.kidney.de/pget.php?&tw=1&pmid=19644062 #FOAvip Background: Transient receptor potential cation channel, subfamily M, member 7 (TRPM7) has been implicated in ischemic brain damage, a major source of morbidity and mortality in westernized society. We hypothesized that the TRPM7 gene variation may play a role in the risk of ischemic stroke. Methods: From a group of DNA samples collected at baseline in a prospective cohort of 14,916 initially healthy American men, we assessed 16 TRPM7 tag-SNPs (dbSNP: rs11854949, rs4775899, rs11635825, rs12905120, rs16973487, rs7173321, rs7163283, rs17520378, rs17520350, rs4775892, rs7174839, rs17645523, rs3109894, rs616256, rs11070795, and rs313158) from 245 Caucasian men who subsequently suffered an incident ischemic stroke, and from 245 age- and smoking-matched Caucasian men who remained free of reported vascular disease during follow-up (controls). Results: All SNPs examined were in Hardy-Weinberg equilibrium. Overall allele, genotype, and haplotype distributions were similar between cases and controls. Marker-by-marker conditional logistic regression analysis, adjusted for potential risk factors, showed no evidence for an association of any of the SNPs tested with ischemic stroke. Further investigation using an Entropy Blocker-defined haplotype-based approach showed similar null findings. Pre-specified analysis limited to participants without baseline diabetes and hypertension (i.e. low-risk group) again showed similar null findings. Conclusions: The present prospective investigation provides no evidence for a role of TRPM7 gene in the risk of incident ischemic stroke. Twit Text FOAVip Gene Variation of the Transient Receptor Potential Cation Channel, Subfamily M, Member 7 (TRPM7) and Risk of Incident Ischemic Stroke: A Prospective, Nested Case-Control Study ????Stroke http://www.kidney.de/pget.php?&tw=1&pmid=19644062 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=19644062 #FOAvip English Wikipedia <ref>{{Cite pmid|19644062}}</ref> Gene Variation of the Transient Receptor Potential Cation Channel, Subfamily M, Member 7 (TRPM7) and Risk of Incident Ischemic Stroke: A Prospective, Nested Case-Control Study #MMPMID19644062 Romero JR; Ridker PM; Zee RY Stroke 2009[Sep]; 40 (9): 2965-8 PMID19644062show ga Background: Transient receptor potential cation channel, subfamily M, member 7 (TRPM7) has been implicated in ischemic brain damage, a major source of morbidity and mortality in westernized society. We hypothesized that the TRPM7 gene variation may play a role in the risk of ischemic stroke. Methods: From a group of DNA samples collected at baseline in a prospective cohort of 14,916 initially healthy American men, we assessed 16 TRPM7 tag-SNPs (dbSNP: rs11854949, rs4775899, rs11635825, rs12905120, rs16973487, rs7173321, rs7163283, rs17520378, rs17520350, rs4775892, rs7174839, rs17645523, rs3109894, rs616256, rs11070795, and rs313158) from 245 Caucasian men who subsequently suffered an incident ischemic stroke, and from 245 age- and smoking-matched Caucasian men who remained free of reported vascular disease during follow-up (controls). Results: All SNPs examined were in Hardy-Weinberg equilibrium. Overall allele, genotype, and haplotype distributions were similar between cases and controls. Marker-by-marker conditional logistic regression analysis, adjusted for potential risk factors, showed no evidence for an association of any of the SNPs tested with ischemic stroke. Further investigation using an Entropy Blocker-defined haplotype-based approach showed similar null findings. Pre-specified analysis limited to participants without baseline diabetes and hypertension (i.e. low-risk group) again showed similar null findings. Conclusions: The present prospective investigation provides no evidence for a role of TRPM7 gene in the risk of incident ischemic stroke. �Gene Variation of the Transient Receptor Potential Cation Channel, Sub(epmc).pdf DeepDyve Pubget Overpricing