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10.1016/j.tcm.2014.07.001 http://scihub22266oqcxt.onion/10.1016/j.tcm.2014.07.001 C4171185!4171185!25112803     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=25112803&cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215 Deprecated: Implicit conversion from float 219.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 219.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 219.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 219.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 219.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 219.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 219.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 253.2 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 253.2 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 253.2 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\25112803.jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       Trends+Cardiovasc+Med 2014 ; 24 (7): 273-8 Nephropedia Template TP {{tp|p=25112803|t=2014. Sinus node dysfunction in catecholaminergic polymorphic ventricular tachycardia ? risk factor and potential therapeutic target?|pdf=|usr=}}{{25112803}} gab.com Text Sinus node dysfunction in catecholaminergic polymorphic ventricular tachycardia risk factor and potential therapeutic target JO: Trends Cardiovasc Med http://www.kidney.de/pget.php?&tw=1&pmid=25112803 #FOAvip Catecholaminergic polymorphic ventricular tachycardia (CPVT) is an inherited rhythm disorder characterized by the occurrence of potentially life-threatening polymorphic ventricular tachyarrhythmias in conditions of physical or emotional stress. The underlying cause is a dysregulation in intracellular Ca handling due to mutations in the sarcoplasmic reticulum Ca release channel. Recent experimental work suggests that the sinus bradycardia that is sometimes observed in CPVT patients may be another primary defect caused by CPVT mutations. Here, we review the pathophysiology of CPVT and discuss the role of sinus node dysfunction as a modulator of arrhythmia risk and potential therapeutic target. Twit Text FOAVip Sinus node dysfunction in catecholaminergic polymorphic ventricular tachycardia risk factor and potential therapeutic target ????Trends Cardiovasc Med http://www.kidney.de/pget.php?&tw=1&pmid=25112803 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=25112803 #FOAvip English Wikipedia <ref>{{Cite pmid|25112803}}</ref> Sinus node dysfunction in catecholaminergic polymorphic ventricular tachycardia ? risk factor and potential therapeutic target? #MMPMID25112803 Faggioni M; van der Werf C; Knollmann B Trends Cardiovasc Med 2014[Oct]; 24 (7): 273-8 PMID25112803show ga Catecholaminergic polymorphic ventricular tachycardia (CPVT) is an inherited rhythm disorder characterized by the occurrence of potentially life-threatening polymorphic ventricular tachyarrhythmias in conditions of physical or emotional stress. The underlying cause is a dysregulation in intracellular Ca handling due to mutations in the sarcoplasmic reticulum Ca release channel. Recent experimental work suggests that the sinus bradycardia that is sometimes observed in CPVT patients may be another primary defect caused by CPVT mutations. Here, we review the pathophysiology of CPVT and discuss the role of sinus node dysfunction as a modulator of arrhythmia risk and potential therapeutic target. �Sinus node dysfunction in catecholaminergic polymorphic ventricular ta(epmc).pdf DeepDyve Pubget Overpricing