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10.1007/s40477-014-0129-y http://scihub22266oqcxt.onion/10.1007/s40477-014-0129-y C4529411!4529411!26261465     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=26261465&cmd=llinks): Failed to open stream: HTTP request failed! 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Characterization of a murine model of cardiorenal syndrome type 1 by high-resolution Doppler sonography |pdf=|usr=}}{{26261465}} gab.com Text Characterization of a murine model of cardiorenal syndrome type 1 by high-resolution Doppler sonography JO: J Ultrasound http://www.kidney.de/pget.php?&tw=1&pmid=26261465 #FOAvip Abstract: Cardiorenal syndrome type 1 (CRS-1) is the acute kidney disfunction caused by an acute worsening of cardiac function. CRS-1 is the consequence of renal vasoconstriction secondary to renin?angiotensin system (RAS) activation. No animal models of CRS-1 are described in literature. Purpose: To characterize a murine model of CRS-1 by using a high-resolution ultrasound echo-color Doppler system (VEVO2100). Materials: Post-ischemic heart failure was induced by coronary artery ligation (LAD) in seven CD1 mice. Fifteen and thirty days after surgery, mice underwent cardiac and renal echo-color Doppler. Serum creatinine and plasma renin activity were measured after killing. Animals were compared to seven CD1 control mice. Results: Heart failure with left ventricle dilatation (end diastolic area, p < 0.05 vs. controls) and significantly reduced ejection fraction (EF; p < 0.01 vs. controls) was evident 15 days after LAD. We measured a significant renal vasoconstriction in infarcted mice characterized by increased renal pulsatility index (PI; p < 0.05 vs. controls) associated to increased creatinine and renin levels (p < 0.05 vs. controls) Conclusions: The mice model of LAD is a good model of CRS-1 evaluable by Doppler sonography and characterized by renal vasoconstriction due to the activation of the renin?angiotensin system secondary to heart failure. Twit Text FOAVip Characterization of a murine model of cardiorenal syndrome type 1 by high-resolution Doppler sonography ????J Ultrasound http://www.kidney.de/pget.php?&tw=1&pmid=26261465 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=26261465 #FOAvip English Wikipedia <ref>{{Cite pmid|26261465}}</ref> Characterization of a murine model of cardiorenal syndrome type 1 by high-resolution Doppler sonography #MMPMID26261465 Pesce P; Cecchetto L; Brocco S; Bolognesi M; Sodhi K; Abraham NG; Sacerdoti D J Ultrasound 2015[Sep]; 18 (3): 229-35 PMID26261465show ga Abstract: Cardiorenal syndrome type 1 (CRS-1) is the acute kidney disfunction caused by an acute worsening of cardiac function. CRS-1 is the consequence of renal vasoconstriction secondary to renin?angiotensin system (RAS) activation. No animal models of CRS-1 are described in literature. Purpose: To characterize a murine model of CRS-1 by using a high-resolution ultrasound echo-color Doppler system (VEVO2100). Materials: Post-ischemic heart failure was induced by coronary artery ligation (LAD) in seven CD1 mice. Fifteen and thirty days after surgery, mice underwent cardiac and renal echo-color Doppler. Serum creatinine and plasma renin activity were measured after killing. Animals were compared to seven CD1 control mice. Results: Heart failure with left ventricle dilatation (end diastolic area, p < 0.05 vs. controls) and significantly reduced ejection fraction (EF; p < 0.01 vs. controls) was evident 15 days after LAD. We measured a significant renal vasoconstriction in infarcted mice characterized by increased renal pulsatility index (PI; p < 0.05 vs. controls) associated to increased creatinine and renin levels (p < 0.05 vs. controls) Conclusions: The mice model of LAD is a good model of CRS-1 evaluable by Doppler sonography and characterized by renal vasoconstriction due to the activation of the renin?angiotensin system secondary to heart failure. äCharacterization of a murine model of cardiorenal syndrome type 1 by h(epmc).pdf DeepDyve Pubget Overpricing