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10.1097/MD.0000000000001179 http://scihub22266oqcxt.onion/10.1097/MD.0000000000001179 C4603008!4603008!26200622     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=26200622&cmd=llinks): Failed to open stream: HTTP request failed! 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Coexistence of Anti-Glomerular Basement Membrane Antibodies and Anti-Neutrophil Cytoplasmic Antibodies in a Child With Human Leukocyte Antigen Susceptibility and Detailed Antibody Description: A Case Report |pdf=|usr=}}{{26200622}} gab.com Text Coexistence of Anti-Glomerular Basement Membrane Antibodies and Anti-Neutrophil Cytoplasmic Antibodies in a Child With Human Leukocyte Antigen Susceptibility and Detailed Antibody Description: A Case Report JO: Medicine (Baltimore) http://www.kidney.de/pget.php?&tw=1&pmid=26200622 #FOAvip Anti-glomerular basement membrane (anti-GBM) disease and anti-neutrophil cytoplasmic antibody (ANCA)-associated vasculitis both could cause rapidly progressive glomerulonephritis. The coexistence of ANCAs and anti-GBM antibodies was known as ?double positive,? which was extremely rare in children. We report a pediatric case with coexistence of ANCAs and anti-GBM antibodies.A 6-year-old girl presented with acute renal failure, hematuria, proteinuria, and oliguria. She was double positive of ANCAs specific to myeloperoxidase, and anti-GBM antibodies. Kidney biopsy confirmed linear immunoglobulin (Ig)G deposit along GBM and 100% of crescent formation in glomeruli; among them 83.3% were cellular crescents. Human leukocyte antigen (HLA) gene typing showed DRB1?1501, an allele strongly associated with anti-GBM disease, and DRB1?0405, an independent risk factor for renal failure in patients with ANCA-associated vasculitis. The titer of anti-GBM antibodies was 1:800, and the predominant IgG subclass was IgG1, which was closely related with severe kidney injury and worse outcome. The target antigen of anti-GBM antibodies was restricted on the noncollagen domain 1 of the ?3 chain of type IV collagen (?3[IV]NC1), with recognitions to both epitopes, EA (?317?31) and EB (?3127?141).This is the first reported pediatric case with coexistence of ANCAs and anti-GBM antibodies, in which the HLA typing and immunologic characters of autoantibodies were identified. The findings on this early-onset patient are meaningful for understanding the mechanisms of both anti-GBM disease and ANCA-associated vasculitis. Twit Text FOAVip Coexistence of Anti-Glomerular Basement Membrane Antibodies and Anti-Neutrophil Cytoplasmic Antibodies in a Child With Human Leukocyte Antigen Susceptibility and Detailed Antibody Description: A Case Report ????Medicine (Baltimore) http://www.kidney.de/pget.php?&tw=1&pmid=26200622 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=26200622 #FOAvip English Wikipedia <ref>{{Cite pmid|26200622}}</ref> Coexistence of Anti-Glomerular Basement Membrane Antibodies and Anti-Neutrophil Cytoplasmic Antibodies in a Child With Human Leukocyte Antigen Susceptibility and Detailed Antibody Description: A Case Report #MMPMID26200622 Xie Lj; Cui Z; Jia Xy; Chen Z; Liu Xr; Zhao Mh Medicine (Baltimore) 2015[Jul]; 94 (29): ä PMID26200622show ga Anti-glomerular basement membrane (anti-GBM) disease and anti-neutrophil cytoplasmic antibody (ANCA)-associated vasculitis both could cause rapidly progressive glomerulonephritis. The coexistence of ANCAs and anti-GBM antibodies was known as ?double positive,? which was extremely rare in children. We report a pediatric case with coexistence of ANCAs and anti-GBM antibodies.A 6-year-old girl presented with acute renal failure, hematuria, proteinuria, and oliguria. She was double positive of ANCAs specific to myeloperoxidase, and anti-GBM antibodies. Kidney biopsy confirmed linear immunoglobulin (Ig)G deposit along GBM and 100% of crescent formation in glomeruli; among them 83.3% were cellular crescents. Human leukocyte antigen (HLA) gene typing showed DRB1?1501, an allele strongly associated with anti-GBM disease, and DRB1?0405, an independent risk factor for renal failure in patients with ANCA-associated vasculitis. The titer of anti-GBM antibodies was 1:800, and the predominant IgG subclass was IgG1, which was closely related with severe kidney injury and worse outcome. The target antigen of anti-GBM antibodies was restricted on the noncollagen domain 1 of the ?3 chain of type IV collagen (?3[IV]NC1), with recognitions to both epitopes, EA (?317?31) and EB (?3127?141).This is the first reported pediatric case with coexistence of ANCAs and anti-GBM antibodies, in which the HLA typing and immunologic characters of autoantibodies were identified. The findings on this early-onset patient are meaningful for understanding the mechanisms of both anti-GBM disease and ANCA-associated vasculitis. äCoexistence of Anti-Glomerular Basement Membrane Antibodies and Anti-N(epmc).pdf DeepDyve Pubget Overpricing