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10.1097/MOG.0000000000000217 http://scihub22266oqcxt.onion/10.1097/MOG.0000000000000217 C4682364!4682364!26398682     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=26398682&cmd=llinks): Failed to open stream: HTTP request failed! 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Crosstalk between type 3 innate lymphoid cells and the gut microbiota in inflammatory bowel disease |pdf=|usr=}}{{26398682}} gab.com Text Crosstalk between type 3 innate lymphoid cells and the gut microbiota in inflammatory bowel disease JO: Curr Opin Gastroenterol http://www.kidney.de/pget.php?&tw=1&pmid=26398682 #FOAvip Purpose of review: Innate lymphoid cells (ILCs) are a newly-identified population of immune cells prevalent in, but not limited to, mucosal tissues that not only play a significant role in immune homeostasis and host defense, but also in disease pathogenesis. This review highlights the importance of type 3 ILCs (ILC3s) and their interactions with the intestinal microflora, both in maintaining gut health and in the development of inflammatory bowel disease (IBD). Recent findings: Distinct lineages of ILCs are defined based on the presence of cell surface proteins, secretion of effector cytokines, and expression of master transcription factors that determine their differentiation and inflammatory behavior. These ILC subgroups mirror corresponding CD4+ T-cell subsets, with which they share many phenotypic, morphologic, and functional attributes. ILC3s, in particular, through direct and indirect interactions with the gut microbiota, have been identified to promote protection and maintenance of epithelial integrity, but also to regulate intestinal inflammation and fibrosis, such as that observed in IBD. Summary: Gut mucosal ILCs respond to environmental cues, such as diet and microflora composition, which can shape downstream immune function. As such, ILCs represent attractive targets for the development of therapeutic modalities to maintain gut health and to potentially treat IBD. Twit Text FOAVip Crosstalk between type 3 innate lymphoid cells and the gut microbiota in inflammatory bowel disease ????Curr Opin Gastroenterol http://www.kidney.de/pget.php?&tw=1&pmid=26398682 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=26398682 #FOAvip English Wikipedia <ref>{{Cite pmid|26398682}}</ref> Crosstalk between type 3 innate lymphoid cells and the gut microbiota in inflammatory bowel disease #MMPMID26398682 Buela KAG; Omenetti S; Pizarro TT Curr Opin Gastroenterol 2015[Nov]; 31 (6): 449-55 PMID26398682show ga Purpose of review: Innate lymphoid cells (ILCs) are a newly-identified population of immune cells prevalent in, but not limited to, mucosal tissues that not only play a significant role in immune homeostasis and host defense, but also in disease pathogenesis. This review highlights the importance of type 3 ILCs (ILC3s) and their interactions with the intestinal microflora, both in maintaining gut health and in the development of inflammatory bowel disease (IBD). Recent findings: Distinct lineages of ILCs are defined based on the presence of cell surface proteins, secretion of effector cytokines, and expression of master transcription factors that determine their differentiation and inflammatory behavior. These ILC subgroups mirror corresponding CD4+ T-cell subsets, with which they share many phenotypic, morphologic, and functional attributes. ILC3s, in particular, through direct and indirect interactions with the gut microbiota, have been identified to promote protection and maintenance of epithelial integrity, but also to regulate intestinal inflammation and fibrosis, such as that observed in IBD. Summary: Gut mucosal ILCs respond to environmental cues, such as diet and microflora composition, which can shape downstream immune function. As such, ILCs represent attractive targets for the development of therapeutic modalities to maintain gut health and to potentially treat IBD. äCrosstalk between type 3 innate lymphoid cells and the gut microbiota (epmc).pdf DeepDyve Pubget Overpricing