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10.1038/srep26777 http://scihub22266oqcxt.onion/10.1038/srep26777 C4880926!4880926!27226113     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=27226113&cmd=llinks): Failed to open stream: HTTP request failed! 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Renal Nitric Oxide Deficiency and Chronic Kidney Disease in Young Sheep Born with a Solitary Functioning Kidney |pdf=|usr=}}{{27226113}} gab.com Text Renal Nitric Oxide Deficiency and Chronic Kidney Disease in Young Sheep Born with a Solitary Functioning Kidney JO: Sci Rep http://www.kidney.de/pget.php?&tw=1&pmid=27226113 #FOAvip Previously, we demonstrated that renal hemodynamic responses to nitric oxide (NO) inhibition were attenuated in aged, hypertensive sheep born with a solitary functioning kidney (SFK). NO is an important regulator of renal function, particularly, in the postnatal period. We hypothesized that the onset of renal dysfunction and hypertension in individuals with a SFK is associated with NO deficiency early in life. In this study, renal and cardiovascular responses to L-NAME infusion (Nw-nitro-L-arginine methyl ester) were examined in 6-month old lambs born with a SFK, induced by fetal unilateral nephrectomy (uni-x). Renal responses to L-NAME were attenuated in uni-x sheep with the fall in glomerular filtration rate (GFR) and urinary sodium excretion (UNaV) being less in the uni-x compared to sham lambs (%?GFR; ?41?�?3?vs ?54?�?4: P?=?0.03, %?UNaV; ?48?�?5?vs ?76?�?3, P?=?0.0008). 24?hour-basal urinary nitrate and nitrite (NOx) excretion was less in the uni-x animals compared to the sham (NOx excretion ?M/min/kg; sham: 57?�?7; uni-x: 38?�?4, P?=?0.02). L-NAME treatment reduced urinary NOx to undetectable levels in both groups. A reduction in NO bioavailability in early life may contribute to the initiation of glomerular and tubular dysfunction that promotes development and progression of hypertension in offspring with a congenital nephron deficit, including those with a SFK. Twit Text FOAVip Renal Nitric Oxide Deficiency and Chronic Kidney Disease in Young Sheep Born with a Solitary Functioning Kidney ????Sci Rep http://www.kidney.de/pget.php?&tw=1&pmid=27226113 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=27226113 #FOAvip English Wikipedia <ref>{{Cite pmid|27226113}}</ref> Renal Nitric Oxide Deficiency and Chronic Kidney Disease in Young Sheep Born with a Solitary Functioning Kidney #MMPMID27226113 Singh RR; Easton LK; Booth LC; Schlaich MP; Head GA; Moritz KM; Denton KM Sci Rep 2016[]; 6 (�): � PMID27226113show ga Previously, we demonstrated that renal hemodynamic responses to nitric oxide (NO) inhibition were attenuated in aged, hypertensive sheep born with a solitary functioning kidney (SFK). NO is an important regulator of renal function, particularly, in the postnatal period. We hypothesized that the onset of renal dysfunction and hypertension in individuals with a SFK is associated with NO deficiency early in life. In this study, renal and cardiovascular responses to L-NAME infusion (Nw-nitro-L-arginine methyl ester) were examined in 6-month old lambs born with a SFK, induced by fetal unilateral nephrectomy (uni-x). Renal responses to L-NAME were attenuated in uni-x sheep with the fall in glomerular filtration rate (GFR) and urinary sodium excretion (UNaV) being less in the uni-x compared to sham lambs (%?GFR; ?41?�?3?vs ?54?�?4: P?=?0.03, %?UNaV; ?48?�?5?vs ?76?�?3, P?=?0.0008). 24?hour-basal urinary nitrate and nitrite (NOx) excretion was less in the uni-x animals compared to the sham (NOx excretion ?M/min/kg; sham: 57?�?7; uni-x: 38?�?4, P?=?0.02). L-NAME treatment reduced urinary NOx to undetectable levels in both groups. A reduction in NO bioavailability in early life may contribute to the initiation of glomerular and tubular dysfunction that promotes development and progression of hypertension in offspring with a congenital nephron deficit, including those with a SFK. �Renal Nitric Oxide Deficiency and Chronic Kidney Disease in Young Shee(epmc).pdf DeepDyve Pubget Overpricing