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10.1007/s13311-016-0430-x http://scihub22266oqcxt.onion/10.1007/s13311-016-0430-x C4965400!4965400!27032406     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=27032406&cmd=llinks): Failed to open stream: HTTP request failed! 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Issues in the Treatment of Neurological Conditions Caused by Reactivation of Varicella Zoster Virus (VZV) |pdf=|usr=}}{{27032406}} gab.com Text Issues in the Treatment of Neurological Conditions Caused by Reactivation of Varicella Zoster Virus (VZV) JO: Neurotherapeutics http://www.kidney.de/pget.php?&tw=1&pmid=27032406 #FOAvip Varicella zoster virus (VZV) is a ubiquitous neurotropic human herpesvirus. Primary infection usually causes varicella (chicken pox), after which virus becomes latent in ganglia along the entire neuraxis. Decades later, virus reactivates to produce herpes zoster (shingles), a painful dermatomally distributed vesicular eruption. Zoster may be further complicated by postherpetic neuralgia, VZV vasculopathy, myelitis, and segmental motor weakness. VZV reactivation has also been associated with giant cell arteritis. This overview discusses treatment of various conditions that often require both corticosteroids and antiviral drugs. Treatment for VZV-associated disease is often based on case reports and small studies rather than large-scale clinical trials. Issues that require resolution include the optimal duration of such combined therapy, more effective treatment for postherpetic neuralgia, whether some treatments should be given orally or intravenously, the widening spectrum of zoster sine herpete, and the role of antiviral therapy in giant cell arteritis.Electronic supplementary material: The online version of this article (doi:10.1007/s13311-016-0430-x) contains supplementary material, which is available to authorized users. Twit Text FOAVip Issues in the Treatment of Neurological Conditions Caused by Reactivation of Varicella Zoster Virus (VZV) ????Neurotherapeutics http://www.kidney.de/pget.php?&tw=1&pmid=27032406 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=27032406 #FOAvip English Wikipedia <ref>{{Cite pmid|27032406}}</ref> Issues in the Treatment of Neurological Conditions Caused by Reactivation of Varicella Zoster Virus (VZV) #MMPMID27032406 Kennedy PGE Neurotherapeutics 2016[Jul]; 13 (3): 509-13 PMID27032406show ga Varicella zoster virus (VZV) is a ubiquitous neurotropic human herpesvirus. Primary infection usually causes varicella (chicken pox), after which virus becomes latent in ganglia along the entire neuraxis. Decades later, virus reactivates to produce herpes zoster (shingles), a painful dermatomally distributed vesicular eruption. Zoster may be further complicated by postherpetic neuralgia, VZV vasculopathy, myelitis, and segmental motor weakness. VZV reactivation has also been associated with giant cell arteritis. This overview discusses treatment of various conditions that often require both corticosteroids and antiviral drugs. Treatment for VZV-associated disease is often based on case reports and small studies rather than large-scale clinical trials. Issues that require resolution include the optimal duration of such combined therapy, more effective treatment for postherpetic neuralgia, whether some treatments should be given orally or intravenously, the widening spectrum of zoster sine herpete, and the role of antiviral therapy in giant cell arteritis.Electronic supplementary material: The online version of this article (doi:10.1007/s13311-016-0430-x) contains supplementary material, which is available to authorized users. �Issues in the Treatment of Neurological Conditions Caused by Reactivat(epmc).pdf DeepDyve Pubget Overpricing