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10.1016/j.neurobiolaging.2016.11.002 http://scihub22266oqcxt.onion/10.1016/j.neurobiolaging.2016.11.002 C5209254!5209254!27940353     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=27940353&cmd=llinks): Failed to open stream: HTTP request failed! 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White matter damage and glymphatic dysfunction in a model of vascular dementia in rats with no prior vascular pathologies |pdf=|usr=}}{{27940353}} gab.com Text White matter damage and glymphatic dysfunction in a model of vascular dementia in rats with no prior vascular pathologies JO: Neurobiol Aging http://www.kidney.de/pget.php?&tw=1&pmid=27940353 #FOAvip We investigated cognitive function, axonal/white matter (WM) changes and glymphatic function of vascular dementia (VaD) using a multiple microinfarction (MMI) model in retired breeder (RB) rats. The MMI model induces significant (p<0.05) cognitive decline that worsens with age starting at 2 weeks, which persists until at least 6 weeks after MMI. RB rats subjected to MMI exhibit significant axonal/WM damage identified by decreased myelin thickness, oligodendrocyte progenitor cell numbers, axon density, synaptic protein expression in the cortex and striatum, cortical neuronal branching, and dendritic spine density in the cortex and hippocampus compared with age matched controls. MMI evokes significant dilation of perivascular spaces as well as water channel dysfunction indicated by decreased Aquaporin-4 (AQP-4) expression around blood vessels. MMI induced glymphatic dysfunction with delayed cerebrospinal fluid (CSF) penetration into the brain parenchyma via paravascular pathways as well as delayed waste clearance from the brain. The MMI model in RB rats decreases AQP-4 and induces glymphatic dysfunction which may play an important role in MMI induced axonal/WM damage and cognitive deficits. Twit Text FOAVip White matter damage and glymphatic dysfunction in a model of vascular dementia in rats with no prior vascular pathologies ????Neurobiol Aging http://www.kidney.de/pget.php?&tw=1&pmid=27940353 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=27940353 #FOAvip English Wikipedia <ref>{{Cite pmid|27940353}}</ref> White matter damage and glymphatic dysfunction in a model of vascular dementia in rats with no prior vascular pathologies #MMPMID27940353 Venkat P; Chopp M; Zacharek A; Cui C; Zhang L; Li Q; Lu M; Zhang T; Liu A; Chen J Neurobiol Aging 2017[Feb]; 50 (ä): 96-106 PMID27940353show ga We investigated cognitive function, axonal/white matter (WM) changes and glymphatic function of vascular dementia (VaD) using a multiple microinfarction (MMI) model in retired breeder (RB) rats. The MMI model induces significant (p<0.05) cognitive decline that worsens with age starting at 2 weeks, which persists until at least 6 weeks after MMI. RB rats subjected to MMI exhibit significant axonal/WM damage identified by decreased myelin thickness, oligodendrocyte progenitor cell numbers, axon density, synaptic protein expression in the cortex and striatum, cortical neuronal branching, and dendritic spine density in the cortex and hippocampus compared with age matched controls. MMI evokes significant dilation of perivascular spaces as well as water channel dysfunction indicated by decreased Aquaporin-4 (AQP-4) expression around blood vessels. MMI induced glymphatic dysfunction with delayed cerebrospinal fluid (CSF) penetration into the brain parenchyma via paravascular pathways as well as delayed waste clearance from the brain. The MMI model in RB rats decreases AQP-4 and induces glymphatic dysfunction which may play an important role in MMI induced axonal/WM damage and cognitive deficits. äWhite matter damage and glymphatic dysfunction in a model of vascular (epmc).pdf DeepDyve Pubget Overpricing