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10.1002/prp2.300 http://scihub22266oqcxt.onion/10.1002/prp2.300 C5368964!5368964!28357126     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=28357126&cmd=llinks): Failed to open stream: HTTP request failed! 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The anti?inflammatory compound palmitoylethanolamide inhibits prostaglandin and hydroxyeicosatetraenoic acid production by a macrophage cell line |pdf=|usr=}}{{28357126}} gab.com Text The anti inflammatory compound palmitoylethanolamide inhibits prostaglandin and hydroxyeicosatetraenoic acid production by a macrophage cell line JO: Pharmacol Res Perspect http://www.kidney.de/pget.php?&tw=1&pmid=28357126 #FOAvip The anti?inflammatory agent palmitoylethanolamide (PEA) reduces cyclooxygenase (COX) activity in vivo in a model of inflammatory pain. It is not known whether the compound reduces prostaglandin production in RAW264.7 cells, whether such an action is affected by compounds preventing the breakdown of endogenous PEA, whether other oxylipins are affected, or whether PEA produces direct effects upon the COX?2 enzyme. RAW264.7 cells were treated with lipopolysaccharide and interferon?? to induce COX?2. At the level of mRNA, COX?2 was induced >1000?fold following 24 h of the treatment. Coincubation with PEA (10 ?mol/L) did not affect the levels of COX?2, but reduced the levels of prostaglandins D2 and E2 as well as 11? and 15?hydroxyeicosatetraenoic acid, which can also be synthesised by a COX?2 pathway in macrophages. These effects were retained when hydrolysis of PEA to palmitic acid was blocked. Linoleic acid?derived oxylipin levels were not affected by PEA. No direct effects of PEA upon the oxygenation of either arachidonic acid or 2?arachidonoylglycerol by COX?2 were found. It is concluded that in lipopolysaccharide and interferon???stimulated RAW264.7 cells, PEA reduces the production of COX?2?derived oxylipins in a manner that is retained when its metabolism to palmitic acid is inhibited. Twit Text FOAVip The anti inflammatory compound palmitoylethanolamide inhibits prostaglandin and hydroxyeicosatetraenoic acid production by a macrophage cell line ????Pharmacol Res Perspect http://www.kidney.de/pget.php?&tw=1&pmid=28357126 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=28357126 #FOAvip English Wikipedia <ref>{{Cite pmid|28357126}}</ref> The anti?inflammatory compound palmitoylethanolamide inhibits prostaglandin and hydroxyeicosatetraenoic acid production by a macrophage cell line #MMPMID28357126 Gabrielsson L; Gouveia?Figueira S; Häggström J; Alhouayek M; Fowler CJ Pharmacol Res Perspect 2017[Apr]; 5 (2): ä PMID28357126show ga The anti?inflammatory agent palmitoylethanolamide (PEA) reduces cyclooxygenase (COX) activity in vivo in a model of inflammatory pain. It is not known whether the compound reduces prostaglandin production in RAW264.7 cells, whether such an action is affected by compounds preventing the breakdown of endogenous PEA, whether other oxylipins are affected, or whether PEA produces direct effects upon the COX?2 enzyme. RAW264.7 cells were treated with lipopolysaccharide and interferon?? to induce COX?2. At the level of mRNA, COX?2 was induced >1000?fold following 24 h of the treatment. Coincubation with PEA (10 ?mol/L) did not affect the levels of COX?2, but reduced the levels of prostaglandins D2 and E2 as well as 11? and 15?hydroxyeicosatetraenoic acid, which can also be synthesised by a COX?2 pathway in macrophages. These effects were retained when hydrolysis of PEA to palmitic acid was blocked. Linoleic acid?derived oxylipin levels were not affected by PEA. No direct effects of PEA upon the oxygenation of either arachidonic acid or 2?arachidonoylglycerol by COX?2 were found. It is concluded that in lipopolysaccharide and interferon???stimulated RAW264.7 cells, PEA reduces the production of COX?2?derived oxylipins in a manner that is retained when its metabolism to palmitic acid is inhibited. äThe anti?inflammatory compound palmitoylethanolamide inhibits prostagl(epmc).pdf DeepDyve Pubget Overpricing