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10.12659/AJCR.903886 http://scihub22266oqcxt.onion/10.12659/AJCR.903886 C5471926!5471926!28596512     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=28596512&cmd=llinks): Failed to open stream: HTTP request failed! 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Paricalcitol as an Antiproteinuric Agent Can Result in the Deterioration of Renal and Heart Function in a Patient with Fabry Disease |pdf=|usr=}}{{28596512}} gab.com Text Paricalcitol as an Antiproteinuric Agent Can Result in the Deterioration of Renal and Heart Function in a Patient with Fabry Disease JO: Am J Case Rep http://www.kidney.de/pget.php?&tw=1&pmid=28596512 #FOAvip Patient: Female, 44Final Diagnosis: Deterioration of renal and heart functionSymptoms: HypercalcemiaMedication: ?Clinical Procedure: LaboratorySpecialty: NephrologyObjective:: Rare disease Background:: Fabry disease is a rare and progressive X-linked inherited disorder of glycosphingolipid metabolism that is due to deficient or absent lysosomal ?-galactosidase A activity. Among its other associated signs and symptoms, patients present with renal failure and proteinuria, which are markers of disease progression. Renin-angiotensin-aldosterone system (RAAS) blockers can slow the progression of chronic renal failure and proteinuria. In fact, some studies have shown the beneficial effects of paricalcitol on proteinuria. Case Report:: We present a case of a female patient with the classic variant of Fabry disease. She was treated with a high dose of paricalcitol as an antiproteinuric agent due to unsatisfactory double-RAAS blockage, which resulted in transient worsening of cardiac and renal function. Conclusions:: Despite the positive effects of paricalcitol as an antiproteinuric agent, as previously shown by some authors, our case highlights the possible serious adverse effects associated with the use of high doses of this drug. Twit Text FOAVip Paricalcitol as an Antiproteinuric Agent Can Result in the Deterioration of Renal and Heart Function in a Patient with Fabry Disease ????Am J Case Rep http://www.kidney.de/pget.php?&tw=1&pmid=28596512 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=28596512 #FOAvip English Wikipedia <ref>{{Cite pmid|28596512}}</ref> Paricalcitol as an Antiproteinuric Agent Can Result in the Deterioration of Renal and Heart Function in a Patient with Fabry Disease #MMPMID28596512 Keber T; Tretjak M; Vujkovac AC; Mravljak M; Ravber K; Vujkovac B Am J Case Rep 2017[]; 18 (ä): 644-8 PMID28596512show ga Patient: Female, 44Final Diagnosis: Deterioration of renal and heart functionSymptoms: HypercalcemiaMedication: ?Clinical Procedure: LaboratorySpecialty: NephrologyObjective:: Rare disease Background:: Fabry disease is a rare and progressive X-linked inherited disorder of glycosphingolipid metabolism that is due to deficient or absent lysosomal ?-galactosidase A activity. Among its other associated signs and symptoms, patients present with renal failure and proteinuria, which are markers of disease progression. Renin-angiotensin-aldosterone system (RAAS) blockers can slow the progression of chronic renal failure and proteinuria. In fact, some studies have shown the beneficial effects of paricalcitol on proteinuria. Case Report:: We present a case of a female patient with the classic variant of Fabry disease. She was treated with a high dose of paricalcitol as an antiproteinuric agent due to unsatisfactory double-RAAS blockage, which resulted in transient worsening of cardiac and renal function. Conclusions:: Despite the positive effects of paricalcitol as an antiproteinuric agent, as previously shown by some authors, our case highlights the possible serious adverse effects associated with the use of high doses of this drug. äParicalcitol as an Antiproteinuric Agent Can Result in the Deteriorati(epmc).pdf DeepDyve Pubget Overpricing